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Record W4226057301 · doi:10.3389/fcell.2022.898569

Editorial: Toll-Like Receptors Throughout Life: From Controlling Physiological Processes to Determinants of Disease

2022· editorial· en· W4226057301 on OpenAlexaff
Francesca Arnaboldi, Silvia Selleri, Michele Sommariva

Bibliographic record

VenueFrontiers in Cell and Developmental Biology · 2022
Typeeditorial
Languageen
FieldImmunology and Microbiology
TopicImmune Response and Inflammation
Canadian institutionsCentre Hospitalier Universitaire Sainte-Justine
Fundersnot available
KeywordsTollReceptorBiologyDiseaseNeuroscienceCell biologyImmunologyMedicineGeneticsInternal medicine

Abstract

fetched live from OpenAlex

From Controlling Physiological Processes to Determinants of DiseaseSince their discovery, Toll-like receptors (TLRs) have been deeply investigated in different areas of the biomedical research.TLRs are usually associated to their ability to trigger a potent immune response against invading pathogens (Kawasaki and Kawai, 2014;Vijay, 2018) and the study by Valdés-López et al. is another proof in this scenario.The Authors described the involvement of the TLR4 signaling as important factor in inducing an antiviral response, mediated by macrophages, against Chikungunya Virus.However, the importance of TLRs is not restricted to the detection of potential harmful microorganisms.It should be recalled that all the mucosal surfaces of human body are virtually colonized by bacteria, the so-called microbiota, and TLRs continuously interact with these commensal microbes, in a bidirectional crosstalk aimed at preserving homeostasis (Semin et al., 2021), as reported in the review by Le Noci et al.By sensing pathogens and commensals, TLRs provide a double beneficial effect either protecting the host and, at the same time, maintaining a good health status.However, the activation of TLRs not always produces a positive outcome and the initiation of a inflammatory process, mediated by TLR signaling pathway, can be associated to the pathogenesis of different diseases, including cancer (Cook et al., 2004).For instance, Zheng et al. investigated the role of TLR9 during the transition from acute kidney injury (AKI) to chronic kidney disease (CKD), demonstrating that TLR9 expressed by macrophages has a critical role in this process.Moreover, it has been observed that bacteria present in the tumor microenvironment trigger TLRs expressed by tumor-infiltrating immune cells but, instead of generating an anti-tumor immune response, promote immunosuppression and sustain cancer cell growth.The impact of TLRs on cancer development and progression has been also evaluated considering the prognostic value of these receptors, as shown in the study by Lu et al.The Authors observed a positive correlation between TLR4 expression and the overall survival of bladder cancer patients.Moreover, low TLR4 expression was also associated to a better response to chemotherapy.Since TLRs can be included in the etiology of several pathological conditions, they can consequently represent potential good targets for novel therapeutic options (El-Zayat et al., 2019).For example, decreasing TLR expression by quercetin treatment was able to reduce the mortality of broilers by impacting on intestinal microbiota composition Ying et al.Exploiting their immune-activation ability, TLRs have been also investigated as immunotherapeutic drugs, alone or in combination with other therapeutic regimens, as comprehensively described by Farooq et al.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.004
metaresearch head score (Gemma)0.013
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: Not applicable
GenreCandidate signal: Editorial · Consensus signal: Editorial
Teacher disagreement score0.022
Threshold uncertainty score0.075

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0040.013
Meta-epidemiology (narrow)0.0050.001
Meta-epidemiology (broad)0.0040.003
Bibliometrics0.0030.001
Science and technology studies0.0020.002
Scholarly communication0.0060.006
Open science0.0040.001
Research integrity0.0110.016
Insufficient payload (model declined to judge)0.0220.017

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.007
GPT teacher head0.239
Teacher spread0.232 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designNot applicable
Domainnot available
GenreEditorial

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations2
Published2022
Admission routes1
Has abstractyes

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