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Record W4246516702 · doi:10.1093/ecco-jcc/jjx002.084

DOP047 Early fibrostenosis in Crohn's disease is associated with multiple susceptibility loci on Immunochip analysis

2017· article· en· W4246516702 on OpenAlexaboutno aff
T. Holvoet, Peter Bossuyt, I. Cleynen, I. De Kock, P. Hindryckx, Séverine Vermeire, D. Laukens, M. De Vos

Bibliographic record

VenueJournal of Crohn s and Colitis · 2017
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicInflammatory Bowel Disease
Canadian institutionsnot available
Fundersnot available
KeywordsInflammatory bowel diseaseMedicineCrohn's diseaseRetrospective cohort studyDiseaseHistopathologyMagnetic resonance imagingInternal medicineGastroenterologyAllelePathologyRadiologyBiologyGeneticsGene

Abstract

fetched live from OpenAlex

Background: Fibrostenosis is a common complication of Crohn's disease (CD) occurring in 30% of patients. Although the pathophysiology of intestinal fibrosis is incompletely understood, evidence suggests a genetic contribution. Previous genetic association and candidate gene studies were based on clinical definitions which lack both sensitivity, specificity and have a high inter-observer disagreement. Additionally, the recent genotype-phenotype analysis by the IIBDGC did not consider the time to development of fibrostenotic disease. As the genetic risk may be more important in patients with early fibrostenosis, we aimed to identify novel genetic markers by focussing on early fibrostenotic disease. Methods: In this multicenter, retrospective nested case-control study computed tomography (CT) and magnetic resonance imaging (MRI) from CD patients obtained between 2002 and 2016 were examined for the presence of ileal fibrostenotic disease. Patients with early fibrostenosis, defined as a the presence of bowel tickening with luminal narrowing and prestenotic dilatation on CT/MRI occurring within 5 years following diagnosis of ileal or ileocolic disease (Montreal L1 or L3), and with available Illumina Immunochip data were included. The control cohort consisted of inflammatory CD patients (Montreal L1 or L3) without arguments for fibrostenotic disease after minimum 10 years follow up. Allelic association was assessed using PLINK v1.07. Results: In total 3024 CT or MRI scans of 2042 CD patients were screened. 112 patients were selected because of fibrostenosis occurring within 5 years of diagnosis. Of these, Immunochip data were available in 60 cases and 49 (82%) had confirmed stenosis on histopathology. 343 inflammatory CD controls with genotype data were included in the analysis. Of the 156.500 SNPs analysed, only rs35223850 in the MIS18BP1 gene passed genome-wide significance level for association with early fibrostenosis. The protein encoding MIS18BP1 is known to bind to the SP1 transcription factor which has been associated with cardiac, liver and kidney fibrosis. Five additional SNPs reached a statistically suggestive significance level of p<5×10–6, including rs116630177 in the IL23R gene, which has previously been associated with systemic sclerosis. Table 1. Identified SNPs associated with early fibrostenosis Conclusions: This carefully phenotyped study reveals an important role for genetic contribution to early development of fibrostenotic complications in CD. Our data suggest a role for MIS18BP1 and the SP1 transcription factor as well as the IL23 pathway in the pathogenesis of early intestinal fibrosis.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.008

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.001
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.008
GPT teacher head0.241
Teacher spread0.233 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations1
Published2017
Admission routes1
Has abstractyes

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