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Record W4285370241 · doi:10.1161/atvb.41.suppl_1.100

Abstract 100: Mitochondrial Dynamics Directs Macrophage Polarization During Atherosclerosis: Implications For Disease Regression

2021· article· en· W4285370241 on OpenAlexaff
Leah I. Susser, My Anh Nguyen, Michèle Geoffrion, Mireille Khacho, Katey J. Rayner

Bibliographic record

VenueArteriosclerosis Thrombosis and Vascular Biology · 2021
Typearticle
Languageen
FieldImmunology and Microbiology
TopicImmune cells in cancer
Canadian institutionsOttawa Heart InstituteUniversity of Ottawa
Fundersnot available
KeywordsMitochondrial fissionMitochondrionMacrophage polarizationCell biologyMacrophagemitochondrial fusionBiologyInflammationMitochondrial ROSPhenotypeMitochondrial DNAImmunologyBiochemistryIn vitro

Abstract

fetched live from OpenAlex

Coronary heart disease is caused by atherosclerosis, a cholesterol-driven inflammatory disease of the vessel wall. A hallmark of atherogenesis is the accumulation of pro-inflammatory (M1) macrophages within the vessel wall, which sustains the inflammatory environment of the atherosclerotic plaque. In contrast, during disease regression, macrophages adopt a pro-resolving, anti-inflammatory phenotype (M2). Macrophage subtypes also exhibit different metabolic programs, where M1 macrophages' preferentiality use glycolytic metabolism and are associated with high levels of reactive oxygen species (ROS), while M2 macrophages primarily use oxidative metabolism. Macrophages adapt to these different metabolic needs and protect from mitochondrial DNA damage by ROS by undergoing cycles of fission (fragmentation) & fusion (elongation), which have been demonstrated to direct differentiation and function in other cell types, including T-cells. We hypothesize that the balance of mitochondrial fusion & fission directs macrophage polarization and may influence macrophage plasticity during atherosclerosis progression and regression. We found that mouse bone marrow-derived macrophages polarized to an M1 phenotype have a more elongated mitochondrial network, whereas both resting (M0) and M2 macrophages have a more fragmented mitochondrial phenotype. We find that M1 macrophages are more active with a higher number of mitochondria undergoing either a fission or fusion event compared to both M0 and M2 cells. The M1-like elongated mitochondrial phenotype can also be seen under atherogenic lipid-loading conditions. Exciting preliminary data suggests inhibiting mitochondrial fission (i.e. by knocking down three critical proteins, Drp1, Mff & Fis1) or inhibiting mitochondrial fusion (i.e. by knocking down fusion proteins Opa1, Mfn1 & Mfn2) impacts macrophages ability to polarizing into M1 or M2 phenotypes. Together these data suggest that the changes in mitochondrial dynamics direct the polarization of these macrophages and thus could be targeted to promote regression. Regression is an untapped area of atherosclerosis research, and mitochondria dynamics may be a new target to direction macrophage polarization within a lesion.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.004
Threshold uncertainty score0.013

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0040.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.025
GPT teacher head0.282
Teacher spread0.257 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2021
Admission routes1
Has abstractyes

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