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Record W4294685605 · doi:10.1093/neuonc/noac174.033

OS02.5.A Alzheimer-type neuropathological changes in glioblastoma-adjacent cortex

2022· article· en· W4294685605 on OpenAlexaff
Lisa Greutter, Yelyzaveta Miller-Michlits, Sigrid Klotz, A Miller-Michlits, Thomas Roetzer-Pejrimovsky, Bernhard Baumann, Barbara Kiesel, Gábor G. Kovács, P Stepper, Adelheid Wöehrer

Bibliographic record

VenueNeuro-Oncology · 2022
Typearticle
Languageen
FieldMedicine
TopicCancer-related cognitive impairment studies
Canadian institutionsUniversity of Toronto
Fundersnot available
KeywordsNeuropathologyPathologyNeuropilMedicineNeurologyCerebral amyloid angiopathyAmyloid (mycology)NeurochemistryInternal medicineDementiaCentral nervous systemDiseasePsychiatry

Abstract

fetched live from OpenAlex

Abstract Background Glioblastoma (GBM) is an aggressive type of brain cancer that is prevalent and fatal in the elderly. Age is not only the most common risk factor for brain cancer but also for neurodegenerative diseases, and previous studies have indicated an excess risk of co-occurrence of both diseases. Here, we aim to map Alzheimer (AD)-related pathology in GBM-adjacent cortex. Material and Methods To this end, we have screened a cohort of 99 individuals with 200 tissue samples comprising tumor and adjacent cortex, including longitudinal samples in 13 patients. The samples were provided by the Division of Neuropathology and Neurochemistry, Medical University of Vienna from 2002 to 2021. Age and tumor location were abstracted from clinical data where available. All samples were stained for A-beta, tau-AT8 and NeuN using immunohistochemistry. Whole slide scans were segmented and protein deposits were quantified with QuPath. Further statistical analyses were conducted with R. Tau pathology was recorded as neurofibrillary tangles, neuropil threads, and astroglial pathology. Likewise, amyloid pathology was assessed as plaques and/or cerebral amyloid angiopathy (CAA). For both proteins, deposits were grouped into: absent, mild, moderate, and severe. Results In the total cohort, median age was 67.5 ys (range 20-92 ys), the female-to-male ratio 0.68. Overall 44.4 % (n=44/99) showed any type of A-beta and/or taupathology, which was strongly correlated with age (R=0.26, p= 0.001). Among them, 38.6 % (n=17/44) had combined pathology, while 36.4 % (n=16/44) displayed pure amyloid-beta, and 25 % (n=11/44) only tau pathology. A-beta pathology comprised plaques in 74.4 % (n=29/39) and CAA in 28.2 % (n=11/29). Consistent with the spatiotemporal evolution of AD, neurofibrillary tangle load was highest in the temporal lobe (42.9 % n=21/49), while plaque load was most prevalent in the occipital lobe (62.5 % n=5/8). This pattern was accentuated in patients above age 65, while it deviated in those below age 65. Over time, the AD-type pathology increased in 38.4 % (n=5/13) and remained stable in 53.8 % (n=7/13) of recurrent tumors. Total cell densities in tumor-infiltrated cortex ranged from 474 to 7,540 cells/mm2, being similar across all lobes. Higher cell density correlated with decreased neuronal counts (R= -0.46, p<0.0001) and decreased AD-load (R= -0.25, p= 0.002). Conclusion Collectively, our results establish frequent co-occurrence of Alzheimer disease neuropathological changes in the GBM-adjacent cortex. They prompt further investigation of shared pathogenic mechanisms and seek to raise awareness for synergistic effects on cognitive decline.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow), Insufficient payload (model declined to judge)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.747
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0000.001
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.001
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.042
GPT teacher head0.324
Teacher spread0.282 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designNot applicable
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2022
Admission routes1
Has abstractyes

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