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VEGF INHIBITION-INDUCED PARP OVERACTIVATION LEADS TO ENDOTHELIAL DYSFUNCTION AND INFLAMMATION: ROLE OF SIRTUIN 1 SIGNALLING

2023· article· en· W4379791219 on OpenAlexaff
Karla B Neves, Rhéure Alves-Lopes, Augusto C. Montezano, Rhian M. Touyz

Bibliographic record

VenueJournal of Hypertension · 2023
Typearticle
Languageen
FieldMedicine
TopicPARP inhibition in cancer therapy
Canadian institutionsMcGill University Health Centre
Fundersnot available
KeywordsMedicineOlaparibPARP inhibitorEndothelial dysfunctionSirtuin 1Cancer researchPoly ADP ribose polymerasePharmacologyInternal medicineEndocrinologyBiologyBiochemistryDownregulation and upregulation

Abstract

fetched live from OpenAlex

Objective: Hypertension is a common unwanted effect of VEGF inhibitors (VEGFi), which are used as anti-angiogenic drugs in cancer treatment. Clinical observations indicate that the magnitude of blood pressure elevation is lower in patients with cancer who are treated with combination VEGFi and olaparib (PARP inhibitor [PARPi]), versus monotherapy. However putative vascular mechanisms are unknown. PARP plays major role in the activation of TRPM2, a redox-sensitive Ca2+ channel, which is associated with hypertension-induced vascular dysfunction. PARP also regulates sirtuin deacetylases activity, especially sirtuin 1 (SIRT1), which is involved in vascular homeostasis and diseases. Here our aim is to investigate whether VEGFi, via PARP activation, leads to vascular dysfunction through disruption of SIRT1 signalling. Design and method: Human aortic endothelial cells (HAEC) and mouse mesenteric arteries were studied. Cells were exposed to axitinib (VEGFi; 1uM) alone or in combination with olaparib (PARPi; 1uM) in the presence or absence of a SIRT1 activator (SRT1720; 2uM). Wire myograph was used to assess vascular function. Results: Axitinib increased PARP activity in a ROS-dependent manner in HAEC (au: [Veh]0.31 vs. [Axi]0.55; [Tiron+Axi] 0.38] whereas SIRT1 activity was reduced by VEGF inhibition (au: [Veh]33760 vs. [Axi]23367), which was prevented by olaparib. This was followed by an increase in expression of acetyl-p53. Activation of SIRT1 decreased levels of MCP-1 and IL-6 and VCAM-1 and ICAM-1 mRNA levels in HAEC exposed to axitinib, which, similarly to olaparib, was accompanied by a reduction in monocytes adhesion to HAEC. U46619- and ET-1-induced vasoconstriction were increased by axitinib, an effect not observed with axitinib plus Olaparib. Pre-incubation with SRT1720 exacerbated the anti-contractile effects of olaparib. Axitinib impaired ACh-induced vasodilation (% relaxation: 70.5 [Ct] vs. 34.8 [Axi]), which was blocked by olaparib and SRT1720. Phosphorylation of the inhibitory site of eNOS was also increased in HAEC (au: [Veh] 0.18 vs. [Axi] 0.37) and restored by SRT1720. Conclusions: Our data indicate that VEGF inhibition-induced PARP overactivation leads to endothelial dysfunction and inflammatory responses via disruption of SIRT1 signalling. We define a putative vasoprotective effect of olaparib that may ameliorate vascular injury induced by VEGFi in cancer treatment.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.010

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.001
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0030.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.041
GPT teacher head0.278
Teacher spread0.237 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2023
Admission routes1
Has abstractyes

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