MétaCan
Menu
Back to cohort
Record W4383554690 · doi:10.3389/fncel.2023.1226630

Preserving and enhancing mitochondrial function after stroke to protect and repair the neurovascular unit: novel opportunities for nanoparticle-based drug delivery

2023· review· en· W4383554690 on OpenAlexafffund
Robyn J. Novorolsky, Gracious D. S. Kasheke, Antoine M. Hakim, Marianna Földvári, Gabriel G. Dorighello, Israel Sekler, Vidyasagar Vuligonda, Martin E. Sanders, Robert Renden, Justin J. Wilson, George S. Robertson

Bibliographic record

VenueFrontiers in Cellular Neuroscience · 2023
Typereview
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicMitochondrial Function and Pathology
Canadian institutionsUniversity of WaterlooDalhousie University
FundersCongressionally Directed Medical Research ProgramsNational Institute of Neurological Disorders and StrokeDalhousie UniversityMultiple Sclerosis Society of CanadaEuropean Genomic Institute for DiabetesHeart and Stroke Foundation of Canada
KeywordsNeurovascular bundleDrug deliveryStroke (engine)DrugFunction (biology)NanoparticleNanotechnologyMedicineNeurosciencePharmacologyBusinessBiomedical engineeringMaterials sciencePsychologySurgeryBiologyEngineeringCell biologyMechanical engineering

Abstract

fetched live from OpenAlex

The neurovascular unit (NVU) is composed of vascular cells, glia, and neurons that form the basic component of the blood brain barrier. This intricate structure rapidly adjusts cerebral blood flow to match the metabolic needs of brain activity. However, the NVU is exquisitely sensitive to damage and displays limited repair after a stroke. To effectively treat stroke, it is therefore considered crucial to both protect and repair the NVU. Mitochondrial calcium (Ca 2+ ) uptake supports NVU function by buffering Ca 2+ and stimulating energy production. However, excessive mitochondrial Ca 2+ uptake causes toxic mitochondrial Ca 2+ overloading that triggers numerous cell death pathways which destroy the NVU. Mitochondrial damage is one of the earliest pathological events in stroke. Drugs that preserve mitochondrial integrity and function should therefore confer profound NVU protection by blocking the initiation of numerous injury events. We have shown that mitochondrial Ca 2+ uptake and efflux in the brain are mediated by the mitochondrial Ca 2+ uniporter complex (MCU cx ) and sodium/Ca 2+ /lithium exchanger (NCLX), respectively. Moreover, our recent pharmacological studies have demonstrated that MCU cx inhibition and NCLX activation suppress ischemic and excitotoxic neuronal cell death by blocking mitochondrial Ca 2+ overloading. These findings suggest that combining MCU cx inhibition with NCLX activation should markedly protect the NVU. In terms of promoting NVU repair, nuclear hormone receptor activation is a promising approach. Retinoid X receptor (RXR) and thyroid hormone receptor (TR) agonists activate complementary transcriptional programs that stimulate mitochondrial biogenesis, suppress inflammation, and enhance the production of new vascular cells, glia, and neurons. RXR and TR agonism should thus further improve the clinical benefits of MCU cx inhibition and NCLX activation by increasing NVU repair. However, drugs that either inhibit the MCU cx , or stimulate the NCLX, or activate the RXR or TR, suffer from adverse effects caused by undesired actions on healthy tissues. To overcome this problem, we describe the use of nanoparticle drug formulations that preferentially target metabolically compromised and damaged NVUs after an ischemic or hemorrhagic stroke. These nanoparticle-based approaches have the potential to improve clinical safety and efficacy by maximizing drug delivery to diseased NVUs and minimizing drug exposure in healthy brain and peripheral tissues.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: none
GenreCandidate signal: Review · Consensus signal: Review
Teacher disagreement score0.978
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.061
GPT teacher head0.271
Teacher spread0.209 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designNot applicable
Domainnot available
GenreReview

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations13
Published2023
Admission routes2
Has abstractyes

Explore more

Same venueFrontiers in Cellular NeuroscienceSame topicMitochondrial Function and PathologyFrench-language works237,207