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Record W4386624016 · doi:10.1093/nar/gkad733

RNF8 ubiquitylation of XRN2 facilitates R-loop resolution and restrains genomic instability in BRCA1 mutant cells

2023· article· en· W4386624016 on OpenAlexafffund
Rehna Krishnan, Mariah Lapierre, Brandon Gautreau, Kevin C. Nixon, Samah El Ghamrasni, Parasvi S. Patel, Jun Hao, V. Talya Yerlici, Kiran Kumar Naidu Guturi, Jonathan St‐Germain, Francesca Mateo, Amine Saad, Arash Algouneh, Rebecca Earnshaw, Duan Shili, Alma Seitova, Joshua W. Miller, Negin Khosraviani, Adam Penn, Brandon Ho, Otto Sánchez, M. Prakash Hande, Jean‐Yves Masson, Grant W. Brown, Moulay A. Alaoui‐Jamali, John J. Reynolds, C.H. Arrowsmith, Brian Raught, Miguel Ángel Pujana, Karim Mekhail, Grant S. Stewart, Anne Hakem, Razqallah Hakem

Bibliographic record

VenueNucleic Acids Research · 2023
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicDNA Repair Mechanisms
Canadian institutionsUniversité LavalStructural Genomics ConsortiumUniversity Health NetworkUniversity of TorontoPrincess Margaret Cancer CentreOntario Tech UniversityMcGill University
FundersStructural Genomics ConsortiumCanadian Institutes of Health ResearchGeneralitat de CatalunyaCancer Research UKCanadian Cancer SocietyFondation du cancer du sein du QuébecCentres de Recerca de CatalunyaUniversity of BirminghamOntario Genomics InstituteWorldwide Cancer ResearchNatural Sciences and Engineering Research Council of CanadaPrincess Margaret Cancer Foundation
KeywordsBiologyGenome instabilitySynthetic lethalityMutantDNA damageCancer researchCarcinogenesisPARP1BRCA2 ProteinCancer cellMolecular biologyGeneticsCell biologyMutationCancerDNAPoly ADP ribose polymeraseGermline mutationGene

Abstract

fetched live from OpenAlex

Breast cancer linked with BRCA1/2 mutations commonly recur and resist current therapies, including PARP inhibitors. Given the lack of effective targeted therapies for BRCA1-mutant cancers, we sought to identify novel targets to selectively kill these cancers. Here, we report that loss of RNF8 significantly protects Brca1-mutant mice against mammary tumorigenesis. RNF8 deficiency in human BRCA1-mutant breast cancer cells was found to promote R-loop accumulation and replication fork instability, leading to increased DNA damage, senescence, and synthetic lethality. Mechanistically, RNF8 interacts with XRN2, which is crucial for transcription termination and R-loop resolution. We report that RNF8 ubiquitylates XRN2 to facilitate its recruitment to R-loop-prone genomic loci and that RNF8 deficiency in BRCA1-mutant breast cancer cells decreases XRN2 occupancy at R-loop-prone sites, thereby promoting R-loop accumulation, transcription-replication collisions, excessive genomic instability, and cancer cell death. Collectively, our work identifies a synthetic lethal interaction between RNF8 and BRCA1, which is mediated by a pathological accumulation of R-loops.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.045
GPT teacher head0.325
Teacher spread0.280 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations27
Published2023
Admission routes2
Has abstractyes

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