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NPY‐Induced Stress Resilience and Downregulation of HCN1 Channels Modifies Expression of Conditioned Fear

2017· article· en· W4389023661 on OpenAlexaff
Maria Bompolaki, J. Amiel Rosenkranz, William F. Colmers, Janice H. Urban

Bibliographic record

VenueThe FASEB Journal · 2017
Typearticle
Languageen
FieldNeuroscience
TopicNeuropeptides and Animal Physiology
Canadian institutionsUniversity of Alberta
FundersJohns Hopkins University
KeywordsDownregulation and upregulationFreezing behaviorFear conditioningBasolateral amygdalaExtinction (optical mineralogy)AmygdalaPsychologyEndocrinologyNeuropeptide Y receptorInternal medicineNeuroscienceMedicineChemistryNeuropeptide

Abstract

fetched live from OpenAlex

The anxiolytic and stress‐buffering properties of neuropeptide Y (NPY) in the basolateral amygdala (BLA) have long been established in animal models as well as in humans. Repeated injections of NPY into the BLA promote long‐lasting resilience to restraint stress (Sadjyk et al ., 2008), which is associated with downregulation of HCN1 channels. In addition to inducing stress resilience, we hypothesize that repeated injections of NPY and downregulation of HCN1 subunits in the BLA will ameliorate freezing behavior in a cued fear conditioning paradigm. Male rats received bilateral infusions of 10pmol NPY or vehicle (100nl; 0.1M PBS) into the BLA repeated daily for 5 days. Two weeks after the injections, animals underwent a cued fear conditioning protocol: On day 1, animals were placed in chamber 1 and received 4 pairings of a tone (1500Hz, 85dB) with a foot shock (0.4mA; fear acquisition). The following day the rats were placed in novel chamber 2 and received 15 trials of the tone without a foot shock at 60s intertrial intervals (fear extinction) and freezing behavior was monitored (Anymaze software). Both vehicle‐ and NPY‐treated rats displayed similar rates and amplitudes of fear acquisition on day 1. On day 2, NPY‐treated rats exhibited significantly lower freezing in the first five sessions of the extinction phase. Additionally, at the time point we performed the fear conditioning experiment, we observed an induction of LC3B expression (an autophagy marker) and downregulation of brain‐derived nerve factor (BDNF), however downregulation of the HCN1 protein is not seen until 4 weeks after the NPY treatment. Lastly, to test the involvement of HCN1 subunits in fear conditioning, HCN1 protein was knocked‐down in the BLA using lentiviral delivery of HCN1‐shRNA. Four weeks after the surgery, the rats underwent the same cued fear conditioning protocol as described above and freezing behavior was monitored. HCN1‐knockdown animals had reduced freezing on the acquisition day. This work indicates that the induction and maintenance of NPY‐induced stress resilience is associated with differential changes in protein expression. NPY‐induced resilience is correlated with BDNF downregulation and induction of autophagic pathways at early time points, while HCN1 downregulation later consolidates a new stress‐relieving baseline in NPY‐treated animals. These findings can be of great importance for the way anxiety disorders like post‐traumatic stress disorder are viewed and treated. Support or Funding Information Work supported by MH090292 (JHU; WFC)

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.004

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.052
GPT teacher head0.292
Teacher spread0.240 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2017
Admission routes1
Has abstractyes

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