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Role of Hypoxia‐Inducible‐Factor‐2α in Transcriptional Regulation of Heme Oxygenase‐1 in Hypoxic Cardiomyocytes

2017· article· en· W4389023744 on OpenAlexafffundabout
Ashley L. Eadie, Melissa A. Allwood, Mathew J. Platt, Jason S. Huber, Purvi Trivedi, Malav N. Madhu, Thomas Pulinilkunnil, Jeremy A. Simpson, Keith R. Brunt

Bibliographic record

VenueThe FASEB Journal · 2017
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicCancer, Hypoxia, and Metabolism
Canadian institutionsUniversity of GuelphSaint John Regional HospitalDalhousie University
FundersNatural Sciences and Engineering Research Council of Canada
KeywordsHeme oxygenaseHMOX1HemeHypoxia (environmental)Hypoxia-inducible factorsHeminChemistryPharmacologyCell biologyBiologyBiochemistryEnzymeOxygenGene

Abstract

fetched live from OpenAlex

INTRODUCTION The heme metabolism pathway is a novel therapeutic target in the management of heart failure, with cytoprotection mediated through induction of heme oxygenase‐1 (HMOX1). However, translation of HMOX1‐targeted therapeutics to the clinic has not occurred. Understanding of heme bioavailability regulation in cardiomyocytes, particularly during hypoxia or heart failure, is incomplete. The molecular response to hypoxia is largely regulated by hypoxia inducible transcription factors (HIFs). HIF1α transcriptionally regulates HMOX1 in acute, severe hypoxia but the role of HIF2α in heme metabolism is unknown. A detailed understanding of the differences in molecular regulation between acute or chronic and severe or moderate hypoxia in cardiomyocytes is required. HYPOTHESIS Hypoxia‐inducible factor 2α (HIF2α) contains heme‐regulatory motifs that are sensitized by heme bioavailability at various degrees of hypoxia in cardiomyocytes to regulate the acute expression of HMOX1 in vitro and in experimental models of heart failure in vivo . METHODS Acute myocardial infarction (AMI)‐induced heart failure was modeled through permanent surgical ligation of the left anterior descending coronary artery in mice. Hypertensive heart failure was modeled through transverse aortic constriction (TAC). Expression of heme regulatory enzymes and hypoxia inducible factors were measured via Western Blot, qPCR and heme content assay. In vitro , hypoxia was established by incubating H9C2 and neonatal rat cardiomyocytes in an adjustable‐oxygen chamber. HIF1α and HIF2α were knocked down or overexpressed through adenoviral vectors. To investigate the role of heme bioavailability in HIF2α sensitization, cells were incubated with the heme surrogate, hemin. RESULTS In vivo , heme levels were increased in both AMI and TAC‐induced heart failure progressively, however not due to increased heme synthesis enzymes. In vitro , HMOX1 was increased by HIF2α more than by HIF1α overexpression. This response was synergistically increased in heme‐replete hypoxia with HIF2α, but not HIF1α, overexpression. CONCLUSIONS We have shown for the first time that HIF2α is a novel regulator of HMOX1. Further understanding of heme homeostasis in the heart is necessary to optimize therapeutic targeting of the heme metabolic pathway in heart failure. Further, we show that HIF2α has a distinct role from HIF1α as it is sensitive to heme bioavailability and not an exclusive oxygen sensor. Support or Funding Information Natural Sciences and Engineering Research Council of Canada

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.006

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.018
GPT teacher head0.250
Teacher spread0.232 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2017
Admission routes3
Has abstractyes

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