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Investigating the Mechanism Of Autoimmune Disease Associated LQTS; anti‐Ro52 IgG Decreases hERG Channel Density

2016· article· en· W4389025931 on OpenAlexaff
John Szendrey, Shetuan Zhang

Bibliographic record

VenueThe FASEB Journal · 2016
Typearticle
Languageen
FieldMedicine
TopicCardiac electrophysiology and arrhythmias
Canadian institutionsQueen's University
Fundersnot available
KeywordshERGPatch clampHEK 293 cellsPotassium channelPharmacologyChemistryIon channelInternal medicineElectrophysiologyMedicineReceptor

Abstract

fetched live from OpenAlex

Background The human ether‐a‐go‐go‐related gene (hERG) is the pore forming subunit of Kv11.1, which is responsible for the rapidly activating delayed rectifier potassium current (I Kr ). Reductions to I hERG through mutations or drug interactions can cause LQTS, which predisposes individuals to ventricular arrhythmia's and sudden cardiac death. hERG has been speculated to be involved in autoimmune disease‐associated LQTS, however the mechanism is poorly understood. In 2006, a Japanese group reported an anti‐Ro52 IgG positive patient with LQTS and accompanying torasdes de pointes. The patient's serum was found to reduce the human ether‐a‐go‐related gene (hERG) channel density in a stable cell line. The authors speculated that anti‐Ro52 underwent a cross reaction with hERG, leading the production of anti‐hERG IgG. Here we investigate in detail this proposed mechanism, and provide strong evidence that anti‐Ro52 IgG reduces hERG channel density and current. Methods Human embryonic kidney (HEK 293) cell lines stably expressing the channel of interest were treated with anti‐Ro52 IgG (Sigma, 4 μg/ml) for 24 h, while saline treated cells served as control. Western blot analysis was used to quantify changes in protein expression, and whole cell patch clamp was performed to quantify changes in current. Results We report that anti‐Ro52 IgG reduced hERG channel density and current. This was unique to hERG in that Kv1.5 and EAG channel density and current were not affected by IgG. Replacing the extracellular S5‐pore linker with that of EAGs instilled immunity to IgG treatment. Furthermore, anti‐Ro52 IgG treatment reduced IKr in rat neonatal cardiomyocytes, significantly increased the APD90, but did not affect other native currents. Our data suggest that IgG decreased hERG membrane expression through inducing channel internalization. Digestion of anti‐Ro52 and isolation of the Fab and Fc fragments revealed that the Fc region of IgG is responsible for reducing hERG. Finally, we are currently testing the effects of IgG positive and negative autoimmune patient serum on the QTc interval of rabbits. Conclusion Our findings provide strong evidence that anti‐Ro52 IgG is responsible for autoimmune disease associated‐LQTS, which mechanistically occurs through a reduction of the hERG current due to IgG‐mediated channel internalization. Support or Funding Information Funded by CIHR and Heart and Stroke

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.005

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.015
GPT teacher head0.239
Teacher spread0.225 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2016
Admission routes1
Has abstractyes

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