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Record W4392394239 · doi:10.3988/jcn.2023.0284

Primary Familial Brain Calcification With <i>XPR1</i> Mutation Presenting With Cognitive Dysfunction

2024· article· en· W4392394239 on OpenAlex

Why this work is in the frame

A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.

aboutThe title or abstract carries a Canadian signal from the geographic lexicon.
no affNo Canadian affiliation: this work is invisible to an affiliation-only frame.
No Canadian affiliation. An affiliation-only frame, the usual design, would never have seen this work. It is one of the works that make the case for inverting the frame.

Bibliographic record

VenueJournal of Clinical Neurology · 2024
Typearticle
Languageen
FieldMedicine
TopicThyroid and Parathyroid Surgery
Canadian institutionsnot available
FundersKorea Health Industry Development Institute
KeywordsCalcificationMedicineMutationCognitionGeneticsInternal medicineBiologyPsychiatryGene

Abstract

fetched live from OpenAlex

Dear Editor,Basal ganglia calcification is a common incidental finding in brain imaging, being reported in 20%-30% of the elderly. 1 More than 50 clinical diagnoses have been reported to be associated with calcium accumulation in the basal ganglia. 2 This condition was previously called familial idiopathic basal ganglia calcification, Fahr syndrome, Fahr's disease, and striopallidodentate calcinosis, but primary familial brain calcification (PFBC) was coined to imply that there is a genetic component.Seven Mendelian genes (SLC20A2, PDGFRB, PDGFB, XPR1, MYORG, JAM2, and CMPK2) for PFBC have been discovered. 3Here we report the first Korean case of PFBC caused by an XPR1 mutation, who presented with early-onset cognitive decline with apathy and mild parkinsonism.A 54-year-old female presented with a 3-year history of cognitive deficit.She complained of difficulty in pronouncing words, comprehending complex sentences, and mathematical calculations, which had progressed over the previous year.She exhibited apathy, depression, anxiety, sleep disturbance, and nocturia.She has been taking medications for hypertension, type-2 diabetes mellitus, and dyslipidemia.There was no family history of any neurological disorders.A neurological examination revealed a moderate degree of dysarthria, mild symmetric bradykinesia, and rigidity in both upper extremities.Her gait and postural stability were intact.She has scores of 10, 17, and 15 on the Unified Parkinson's Disease Rating Scale part III score, Mini-Mental State Examination (MMSE), and Montreal Cognitive Assessment Test (MoCA), respectively.A comprehensive neuropsychological study using the Seoul Neuropsychological Screening Battery revealed mild cognitive impairment involving multiple cognitive domains including memory, attention, language, and visuospatial function.Her score on the Korean version of the Short Form of the Geriatric Depression Scale (SGDS-K) was 9, suggesting mild depression.Her score on the Withdrawal/Apathy/Lack of Vigor (WAV) subscale was 3/3.Laboratory studies revealed normal levels of serum calcium, inorganic phosphorous, 25-hydroxy vitamin D, osteocalcin, and parathyroid hormone.Low-dose methimazole was started since a thyroid function test indicated subclinical hypothyroidism.Other routine blood test results were normal.Computed tomography (Fig. 1A-C) and magnetic resonance imaging (MRI) (Fig. 1E-H) revealed dense calcifications in both corona radiata, the basal ganglia (especially the globus pallidus), and the dentate nucleus of the cerebellum. 18F-FP-CIT [(3-[ 18 F]fluoropropyl)-2carbon ethoxy-3-(4-iodophenyl) nortropane] positron-emission tomography (PET) showed preserved dopamine transporter (DAT) binding (Fig. 1D).Fluorodeoxyglucose (FDG) PET showed diffusely decreased FDG uptake in the frontal and parietal lobes and the cerebellum, while showing relatively preserved uptake in the sensorimotor cortex and basal ganglia (Fig. 1I-L).Screening for mutations by whole-exome sequencing revealed the heterozygous missense mutation c.1871G>A (p.Arg624His, NM_004736.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.001
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.248
Threshold uncertainty score0.454

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.041
GPT teacher head0.358
Teacher spread0.317 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it