MétaCan
Menu
Back to cohort
Record W4396991573 · doi:10.1681/asn.20213210s1429a

A Human Missense Integrin-Linked Kinase Variant Negatively Regulates Murine Renal Branching Morphogenesis via mTOR Signaling

2021· article· en· W4396991573 on OpenAlexaff
Xiangyue Hu, Dana Kablawi, Kirsten Y. Renkema, Nine V.A.M. Knoers, Norman D. Rosenblum

Bibliographic record

VenueJournal of the American Society of Nephrology · 2021
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicRenal and related cancers
Canadian institutionsHospital for Sick ChildrenUniversity of Toronto
Fundersnot available
KeywordsMissense mutationIntegrinCell biologyMorphogenesisPI3K/AKT/mTOR pathwaySignal transductionKinaseBiologyCancer researchGeneGeneticsReceptorMutation

Abstract

fetched live from OpenAlex

Background: Branching morphogenesis is critical to kidney development and the pathogenesis of Congenital Anomalies of the Kidney and Urinary Tract (CAKUT). Identification of gene variants via genomic sequencing aims to elucidate molecular mechanisms underlying CAKUT. The pathogenic contributions of such variants are largely unknown; functional analyses are required to identify pathogenic mechanisms. Here, we identify pathogenic effects of a CAKUT-associated human missense variant of Integrin-Linked Kinase (ILK), a key regulator of renal branching morphogenesis, on ureteric branching. Methods: Targeted gene panel sequencing was performed to identify gene variants. ILK-T173I function was investigated in mouse inner medullary collecting duct (mIMCD3) cells and mouse embryonic kidney explants transduced with lentivirus expressing ILK-T173I. Gene expression was analyzed by RNA microarray and validated by qPCR and Western analysis. Mutant mice with a ILK-c.518C>T point mutation were generated using CRISPR/Cas9. Morphogenic effects of ILK-T173I on ureteric branching were visualized using Hoxb7-driven fluorescent marker (MyrVenus) and quantitated by counts of ureteric bud tips and nephrons. Results: An ILK missense variant, ILK-T173I, was identified in a CAKUT patient and her mother by targeted gene panel sequencing and verified by Sanger sequencing. mIMCD3 cells expressing ILK-T173I demonstrated dysregulated expression of AKT/mTOR target mRNAs, identified by RNA microarray and qPCR, and elevated levels of phospho-p70-S6Kinase, a mTOR target (n=3, P=0.03). Overexpression of ILK-T173I in embryonic kidney explants increased phospho-p70-S6Kinase expression (n=3, P=0.03) and decreased ureteric tip number by 50% (n=15, P=0.003), both of which were rescued by treatment with Rapamycin, an mTOR inhibitor (n=4, P=0.04). Knock-in mice in which ILK-T173I replaces the ILK-WT allele were characterized by low nephron number (n=6, P=0.04) and decreased ureteric branching (n=5, P=0.006), and increased expression of phospho-p70-s6Kinase (n=3, P=0.014). Treatment of mutant cultured embryonic kidney explants with Rapamycin rescued ureteric branching to levels observed in Ilk-WT mice. Conclusions: Human Ilk-T173I variant decreases branching morphogenesis in a mTOR-dependent manner. Increased mTOR signaling disrupts mouse kidney development. Funding: Government Support - Non-U.S.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.006

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.010
GPT teacher head0.246
Teacher spread0.237 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2021
Admission routes1
Has abstractyes

Explore more

Same venueJournal of the American Society of NephrologySame topicRenal and related cancersFrench-language works237,207