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Record W4397041297 · doi:10.1681/asn.20233411s1494a

Cell Surface GRP78 and α2M* Are Important Mediators of Tubulointerstitial Fibrosis in CKD

2023· article· en· W4397041297 on OpenAlexaff
Jackie Trink, Melissa Macdonald, Bo Gao, Joan C. Krepinsky

Bibliographic record

VenueJournal of the American Society of Nephrology · 2023
Typearticle
Languageen
FieldMedicine
TopicCystic Fibrosis Research Advances
Canadian institutionsMcMaster University
Fundersnot available
KeywordsFibrosisMedicineKidney diseaseInternal medicineUrology

Abstract

fetched live from OpenAlex

Background: Diabetic kidney disease (DKD) is characterized by glomerular accumulation of extracellular matrix (ECM) proteins followed by the development of tubulointerstitial fibrosis. We recently showed the endoplasmic reticulum resident GRP78 translocates to the cell surface (csGRP78) in response to HG, promoting profibrotic responses in mesangial cells. We further implicated activated alpha 2 macroglobulin (α2M*) as an activator of csGRP78 signaling. Based on increased expression of this receptor-ligand pair in both DKD and non-diabetic chronic kidney disease (CKD) mouse models, we thus wanted to elucidate this signaling pathway's influence on other cell types relevant to kidney disease (proximal tubule epithelial cells (PTEC) and renal fibroblasts (RF)) as well as a potential role for TGFβ1-induced signaling. Methods: PTEC and RF were treated with 30mM HG or 5ng/mL TGFβ1 plus csGRP78 or α2M* inhibitors. Standard molecular biology techniques were used for assessment. Immunohistochemistry staining was conducted on kidney tissues from mouse models of DKD and CKD. Results: We observed increased localization of GRP78 to the surface of PTEC and RF with either HG or TGFβ1 stimulus. Further, α2M expression and activation were shown to be increased by both HG and TGFβ1. Inhibition of either csGRP78 or α2M* prevented HG and TGFβ1-induced ECM production (fibronectin and collagen IV). By HG treatment, downstream TGFβ1 signaling (measured by activation of Smad3) was attenuated by csGRP78 or α2M* inhibition. Interestingly, we observed no effect on Smad3 activation with csGRP78 or α2M* inhibition with TGFβ1 treatment. We hypothesized a potential role for non-canonical TGFβ1 signaling being mediated by csGRP78/ α2M*. We next assessed the known non-canonical TGFβ1 signaling molecules yes associated protein (YAP) and transcriptional co-activator with PDZ-binding motif (TAZ). Here we observed that inhibition of either csGRP78 and α2M* attenuated YAP and TAZ expression under both HG and TGFβ1 treatment. This implicates Smad-dependent and independent signaling being mediated by csGRP78/ α2M*. Conclusions: These data support a role for csGRP78/α2M* in mediating HG or TGFβ1-induced profibrotic signaling in PTEC and RF. Inhibition of this signaling pathway represents a novel target for preventing DKD or CKD-associated fibrosis which we are currently evaluating in vivo. Funding: Government Support - Non-U.S.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.004

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.012
GPT teacher head0.292
Teacher spread0.281 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2023
Admission routes1
Has abstractyes

Explore more

Same venueJournal of the American Society of Nephrology→Same topicCystic Fibrosis Research Advances→French-language works237,207→