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Record W4397046221 · doi:10.1681/asn.20223311s1429c

Role of P2X Receptors on Endothelial Cell Injury Mediated by Complement Activation

2022· article· en· W4397046221 on OpenAlexaffabout
Arlette Bohorquez, Carolina G. Ortiz-Sandoval, Daniel Diatlov, Christoph Licht

Bibliographic record

VenueJournal of the American Society of Nephrology · 2022
Typearticle
Languageen
FieldMedicine
TopicRenal Diseases and Glomerulopathies
Canadian institutionsHospital for Sick Children
Fundersnot available
KeywordsReceptorComplement systemCell biologyComplement (music)Endothelial stem cellImmunologyChemistryBiologyMedicineInternal medicineAntibodyBiochemistryPhenotype

Abstract

fetched live from OpenAlex

Background: The complement system is critical for to innate immunity. The complement system consists of over 50 proteins, provides defense against microbes, mediates inflammatory responses. The final step of complement activation is formation of the membrane attack complex (MAC, C5b-9). Complement over-activation is implicated in the pathophysiology of numerous diseases, yet the mechanisms underlying host cell damage are not fully elucidated. The P2X receptors, are transmembrane cationic channels gated by adenosine triphosphate (ATP) which are present in the plasma membrane (PM) of most excitable and non-excitable cells including all segments of the nephron, and renal cells. Recently, a link between complement system and P2X receptors has been established, showing that inhibition of P2X activation decreases complement mediated cell damage. The aim of this study is to determine the role of P2X receptors activity in complement activation on primary endothelial cells. Methods: Complement was activated on blood outgrowth endothelial cells (BOECs) using an established protocol, first sensitizing the cells using a specific antibody to CD59, followed by treatment with normal human serum. Complement activity was assessed by measuring the deposition of C3b and C5b-9 on BOEC PM via immunofluorescence using specific antibodies. Intracellular Ca2+ levels were measured using a fluorescent calcium indicator (Invitrogen). ATP release was measured using a commercial ATP luminescence assay (Promega). Results: Complement activation caused C3b and C5b-9 deposition on the PM of BOECs, followed by Ca2+ influx and ATP release. Cells treated with P2X receptor antagonists, showed a significant decrease in C5b-9 deposition compared to untreated controls. In addition, P2X antagonist treatment significantly ameliorated Ca2+ influx as well as ATP release. Conclusions: The finding of reduced C5b-9 deposition on BOECs in the presence of P2X receptor antagonists suggests an important functional link between the complement system and purinergic system. While more research is required to fully elucidate the interactions between these critical, ubiquitous biological systems, our results contribute to a better understanding of the consequences of complement activation on endothelial cells and suggest new therapeutic targets for disease related to dysregulated complement system. Funding: Commercial Support - Paragon Ventures Inc, Vancouver, BC, Canada

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.010

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0030.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.009
GPT teacher head0.261
Teacher spread0.252 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2022
Admission routes2
Has abstractyes

Explore more

Same venueJournal of the American Society of Nephrology→Same topicRenal Diseases and Glomerulopathies→French-language works237,207→