MétaCan
Menu
Back to cohort
Record W4402131442 · doi:10.1016/j.cccb.2024.100318

TREM2-regulated microglial reactivity confers resilience against vascular contributions to white matter injury and cognitive decline

2024· article· en· W4402131442 on OpenAlexaff
Stefan Szymkowiak, Mila Redzic, Anirudh Patir, Clare Latta, Michael J. Daniels, Jack Barrington, Katie Askew, Jessica Duncombe, Karen Horsburgh, Barry W. McColl

Bibliographic record

VenueCerebral Circulation - Cognition and Behavior · 2024
Typearticle
Languageen
FieldNeuroscience
TopicNeuroinflammation and Neurodegeneration Mechanisms
Canadian institutionsDiscovery Centre
Fundersnot available
KeywordsWhite matterMicrogliaTREM2Cognitive declineResilience (materials science)NeuroscienceReactivity (psychology)CognitionPsychologyMedicineInflammationInternal medicinePathologyDementiaPhysicsMagnetic resonance imaging

Abstract

fetched live from OpenAlex

Cerebrovascular disease is a major contributor to subcortical white matter pathology, vascular cognitive impairment (VCI) and dementia. Although the precise pathophysiological mechanisms remain unclear, it is increasingly evident that microglial responses may be critical to the development and progression of cerebrovascular disease and its cognitive consequences. To investigate this further, we assessed microglial reactivity in white matter regions of human post- mortem tissue with pathologically confirmed cerebrovascular injury. In parallel, we implemented a mouse model of VCI induced by bilateral carotid artery stenosis (BCAS) and determined the impact of genetically deleting the microglial immunoreceptor triggering receptor expressed on myeloid cells 2 (TREM2), a key regulator of microglial homeostatic and reactive functions. Using immunohistochemistry (IHC) and quantitative polymerase chain reaction (QPCR) we observed increased microglial reactivity and TREM2 expression in our human cohort implicating TREM2 signalling in microglial responses to cerebrovascular. In mice, IHC and flow cytometric analysis revealed Trem2 deficiency blunted microglial reactivity following BCAS (1 month). Transcriptomic analysis of Trem2-/- microglia also demonstrated attenuated induction of gene expression modules associated with inflammation, lysosomal function, lipid processing and metabolic reprogramming following BCAS (data not shown). Importantly, this was associated with greater white matter injury compared to wildtype (WT) counterparts in the absence of overt cognitive changes. Following longer durations of BCAS (6 months), Trem2-/- mice exhibited additional parenchymal and vascular pathologies associated with impairments in spatial learning and memory whilst such changes were largely absent in WT mice. Interestingly, assessment of microglia by IHC demonstrated reduced interaction with white matter cerebrovasculature in Trem2-/- mice. Taken together, these data support a role for microglia in cerebrovascular contributions to cognitive decline and dementia. Therefore, targeting regulators of microglia reactivity may provide means to influence cerebrovascular disease trajectory and associated cognitive changes. While further work is required to delineate the precise function of microglia in this context, our preclinical data suggest TREM2-regulated microglial responses to cerebrovascular insult are important for maintenance of brain health and cognitive resilience. Future work will continue to decipher how TREM2 mediates such resilience with particular focus on interaction with other cell types within the neurogliovascular unit.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.178
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.001
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.020
GPT teacher head0.292
Teacher spread0.272 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2024
Admission routes1
Has abstractyes

Explore more

Same venueCerebral Circulation - Cognition and BehaviorSame topicNeuroinflammation and Neurodegeneration MechanismsFrench-language works237,207