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Record W4402609603 · doi:10.1101/2024.09.15.613131

Off-Equilibrium Fluctuation-Dissipation Theorem Paves the Way in Alzheimer’s Disease Research

2024· preprint· en· W4402609603 on OpenAlexfundno aff
Gustavo Patow, J M Monti, Irene Acero-Pous, Sebastián Idesis, Anira Escrichs, Yonatan Sanz Perl, Petra Ritter, Morten L. Kringelbach, Gustavo Deco

Bibliographic record

VenuebioRxiv (Cold Spring Harbor Laboratory) · 2024
Typepreprint
Languageen
FieldPhysics and Astronomy
TopicAdvanced Thermodynamics and Statistical Mechanics
Canadian institutionsnot available
FundersAgencia Estatal de InvestigaciónCanadian Institutes of Health ResearchNational Institutes of HealthGenentechIXICOH. Lundbeck A/SServierEisaiNorthern California Institute for Research and EducationPfizerNovartis Pharmaceuticals CorporationUniversity of Southern CaliforniaBiogenEli Lilly and CompanyBristol-Myers SquibbBioClinicaEuropean Regional Development FundU.S. Department of DefenseAlzheimer's Disease Neuroimaging InitiativeMeso Scale DiagnosticsAlzheimer's Association
KeywordsFluctuation-dissipation theoremDissipationStatistical physicsPhysicsDiseaseMathematical economicsMathematicsMedicineQuantum mechanicsInternal medicine

Abstract

fetched live from OpenAlex

ABSTRACT INTRODUCTION Alzheimer’s disease (AD) is a neurodegenerative disorder characterized by progressive cognitive decline. Although traditional methods have provided insights into brain dynamics in AD, they have limitations in capturing non-equilibrium dynamics across disease stages. Recent studies suggest that dynamic functional connectivity in resting-state networks (RSNs) may serve as a biomarker for AD, but the role of deviations from dynamical equilibrium remains underexplored. OBJECTIVE This study applies the off-equilibrium fluctuation-dissipation theorem (FDT) 1 to analyze brain dynamics in AD, aiming to compare deviations from equilibrium in healthy controls, patients with mild cognitive impairment (MCI), and those with AD. The goal is to identify potential biomarkers for early AD detection and understand disease progression’s mechanisms. METHODS We employed a model-free approach based on FDT to analyze functional magnetic resonance imaging (fMRI) data, including healthy controls, MCI patients, and AD patients. Deviations from equilibrium in resting-state brain activity were quantified using fMRI scans. In addition, we performed model-based simulations incorporating Amyloid-Beta (A β ), tau burdens, and Generative Effective Connectivity (GEC) for each subject. RESULTS Our findings show that deviations from equilibrium increase during the MCI stage, indicating hyperexcitability, followed by a significant decline in later stages of AD, reflecting neuronal damage. Model-based simulations incorporating A β and tau burdens closely replicated these dynamics, especially in AD patients, highlighting their role in disease progression. Healthy controls exhibited lower deviations, while AD patients showed the most significant disruptions in brain dynamics. DISCUSSION The study demonstrates that the off-equilibrium FDT framework can accurately characterize brain dynamics in AD, providing a potential biomarker for early detection. The increase in non-equilibrium deviations during the MCI stage followed by their decline in AD offers a mechanistic explanation for disease progression. Future research should explore how combining this framework with other dynamic brain measures could further refine diagnostic tools and therapeutic strategies for AD and other neurodegenerative diseases.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Theoretical or conceptual · Consensus signal: Theoretical or conceptual
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.500
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.001
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0010.001
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.027
GPT teacher head0.293
Teacher spread0.266 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designTheoretical or conceptual
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations3
Published2024
Admission routes1
Has abstractyes

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