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Record W4402635937 · doi:10.1101/2024.09.14.613072

<i>Cnes2b</i> Regulates Host Resistance, Inflammatory Responses and Tissue Damage Following <i>Cryptococcus deneoformans</i> Infection

2024· preprint· en· W4402635937 on OpenAlexafffund
Isabelle Angers, Annie Beauchamp, Marwa El-Sheikh, Eva Kaufmann, Donald C. Vinh, Salman T. Qureshi

Bibliographic record

VenuebioRxiv (Cold Spring Harbor Laboratory) · 2024
Typepreprint
Languageen
FieldMedicine
TopicFungal Infections and Studies
Canadian institutionsQueen's UniversityMcGill UniversityChristie (Canada)McGill University Health Centre
FundersCanadian Institutes of Health ResearchMcGill University Health CentreMcGill University
KeywordsHost resistanceHost (biology)MicrobiologyInflammationCryptococcusImmunologyBiologyImmune systemGenetics

Abstract

fetched live from OpenAlex

Abstract The 32.1 Mb Cnes2 chromosome 17 interval was shown to confer resistance to progressive Cryptococcus deneoformans 52D infection. To refine the location of Cnes2 host resistance genes, a subcongenic mouse strain (B6.CBA- Cnes2b ) that contains 8.7 Mb from the telomeric region of Cnes2 was created. At 28 days postinfection B6.CBA- Cnes2b mice had a lower lung fungal burden, increased lung injury, as well as mortality compared to C57BL/6N. B6.CBA- Cnes2b mice had increased pulmonary production of pro-inflammatory mediators, chemokines and Th1-type cytokines as well as increased recruitment of monocytes and neutrophils to the lungs. Cnes2b also regulated several elements of the host response to C. deneoformans 52D infection in a sex-dependent manner. Specifically, male B6.CBA- Cnes2b mice had a lower lung fungal burden, increased brain injury and mortality relative to females. Taken together these findings demonstrate that Cnes2b regulates host inflammation in a manner that controls fungal burden and increases tissue damage. Precise identification of the genes encoded by Cnes2b could reveal key mechanisms of cryptococcal host resistance and immune reconstitution or postinfectious inflammatory syndromes. Importance The 32.1 Mb Cnes2 congenic interval from chromosome 17 of resistant CBA/J mice regulates host resistance to C. deneoformans 52D infection. This study characterizes the host response of B6.CBA- Cnes2b mice that carry an 8.7 Mb sub-congenic interval derived from Cnes2 following C. deneoformans 52D infection. B6.CBA- Cnes2b mice had reduced lung fungal burden, increased lung and brain injury, and mortality. The effects of Cnes2b differed between male and female subcongenic mice and are consistent with known sex differences in human cryptococcal disease. The host response of B6.CBA- Cnes2b mice reflects a crucial balance between effective control of fungal burden and potentially deleterious consequences of enhanced inflammation during cryptococcal infection as predicted by the damage response framework. Further analysis of the Cnes2b sub-congenic interval will lead to definitive identification of genes that confer resistance to progressive cryptococcal infection and/or contribute to deleterious inflammatory responses. Defining key mechanisms that regulate the immune response to Cryptococcus sp. is an important step towards the development of host-directed therapeutics that could improve disease outcomes.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.006

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.010
GPT teacher head0.241
Teacher spread0.231 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2024
Admission routes2
Has abstractyes

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