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Record W4402983755 · doi:10.1101/2024.09.29.24314328

Distribution of White Matter Hyperintensities across Arterial Territories in Neurodegenerative Diseases

2024· preprint· en· W4402983755 on OpenAlexafffundabout
Ikrame Housni, Flavie E. Detcheverry, Manpreet Singh, Mahsa Dadar, Chloe Anastassiadis, Ali Filali‐Mouhim, Mario Masellis, Zahinoor Ismail, Eric E. Smith, Simon Duchesne, Maria Carmela Tartaglia, Natalie A. Phillips, Sridar Narayanan, AmanPreet Badhwar

Bibliographic record

VenuemedRxiv · 2024
Typepreprint
Languageen
FieldNeuroscience
TopicNeurological Disease Mechanisms and Treatments
Canadian institutionsMontreal Neurological Institute and HospitalUniversity Health NetworkUniversity of CalgaryMcGill UniversityDouglas Mental Health University InstituteUniversité LavalUniversité de MontréalSunnybrook Health Science CentreInstitut Universitaire de Gériatrie de Montréal
FundersFonds de Recherche du Québec - SantéNational Institutes of HealthCourtois FoundationConsortium canadien en neurodégénérescence associée au vieillissementCanadian Institutes of Health ResearchUniversity of Southern CaliforniaUniversity of WashingtonUniversity of California, San FranciscoMassachusetts General Hospital
KeywordsHyperintensityWhite matterMedicineDistribution (mathematics)CardiologyInternal medicineMagnetic resonance imagingRadiologyMathematics

Abstract

fetched live from OpenAlex

ABSTRACT MRI-detected white matter hyperintensities (WMH) are often recognized as markers of cerebrovascular abnormalities and an index of vascular brain injury. The literature establishes a strong link between WMH burden and cognitive decline, and suggests that the anatomical distribution of WMH mediates cognitive dysfunction. Pathological remodeling of major cerebral arteries (anterior, ACA; middle, MCA; posterior, PCA) may increase WMH burden in an arterial territory (AT)-specific manner. However, this has not been systematically studied across neurodegenerative diseases (NDDs). To address this gap, we aimed to assess WMH distribution (i) across ATs per clinical category, (ii) across clinical categories per AT, and (iii) between men and women. We also investigated the association between AT-specific WMH burden and cognition. Using two cohorts – Canadian CCNA-COMPASS-ND (N=927) and US-based NIFD (N=194) – we examined WMH distribution across ten clinical categories: cognitively unimpaired (CU), subjective cognitive decline (SCD), mild cognitive impairment (MCI), Alzheimer disease (AD), MCI and AD with high vascular injury (+V), Lewy body dementia, frontotemporal dementia, Parkinson’s disease (PD), and PD with cognitive impairment or dementia. WMH masks were segmented from FLAIR MRI and mapped onto an arterial atlas. Cognitive performance was assessed using four psychometric tests evaluating reaction time and overall cognition, namely Simple Reaction Time (SRT), Choice Reaction Time (CRT), Digit Symbol Substitution Test (DSST), and Montreal Cognitive Assessment (MoCA). Statistical analyses involved linear regression models, controlling for demographic factors, with a 5% False Discovery Rate for multiple comparisons. Our transdiagnostic analysis revealed unique AT-specific WMH burden patterns. Comparisons between ACA and PCA territories revealed distinct burden patterns in clinical categories with similar whole-brain WMH burden, while the MCA territory consistently exhibited the highest burden across all categories, despite accounting for AT size. Hemispheric asymmetries were noted in seven diagnostic categories, with most showing higher WMH burden in the left MCA territory. Our results further revealed distinct AT-specific WMH patterns in diagnostic groups that are more vascular than neurodegenerative (i.e., MCI+V, AD+V). Categories often misdiagnosed in clinical practice, such as FTD and AD, displayed contrasting WMH signatures across ATs. SCD showed distinct AT-specific WMH patterns compared to CU and NDD participants. Additionally, sex-specific differences emerged in five NDDs, with varying AT effects. Importantly, AT-specific WMH burden was associated with slower processing speed in MCI (PCA) and AD (ACA, MCA). This study highlights the importance of evaluating WMH distribution through a vascular-based brain parcellation. We identified ATs with increased vulnerability to WMH accumulation across NDDs, revealing distinct WMH signatures for multiple clinical categories. In the AD continuum, these signatures correlated with cognitive impairment, underscoring the potential for vascular considerations in imaging criteria to improve diagnostic precision.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.068
Threshold uncertainty score0.136

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0020.002
Science and technology studies0.0010.000
Scholarly communication0.0010.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.027
GPT teacher head0.279
Teacher spread0.252 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2024
Admission routes3
Has abstractyes

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