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Record W4404112507 · doi:10.26685/urncst.706

The Impact of Extracellular Vesicles on Inflammation in the Tumor Microenvironment of Sarcomas: A Literature Review

2024· review· en· W4404112507 on OpenAlexaff
Bob-Shen Yan, Ethan J. Schmidt

Bibliographic record

VenueUndergraduate Research in Natural and Clinical Science and Technology (URNCST) Journal · 2024
Typereview
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicExtracellular vesicles in disease
Canadian institutionsMcMaster University
Fundersnot available
KeywordsExtracellular vesiclesInflammationTumor microenvironmentVesicleCancer researchExtracellularMicrovesiclesChemistryBiologyCell biologyTumor cellsImmunologyBiochemistrymicroRNAMembraneGene

Abstract

fetched live from OpenAlex

Introduction: Sarcomas are a diverse group of tumours encompassing over 100 different subtypes occurring in bone, muscle, and cartilage. The sarcoma tumour microenvironment (TME) contains various immune modulators and is influenced by the specific sarcoma subtype. The sarcoma TME is modulated by secretion of cytokines, chemokines, and extracellular vesicles (EVs). EVs play a role in inflammatory and immune responses in the TME. EVs effects are attributed to their cargo, consisting of mRNAs, proteins, miRNAs, chemokines, and cytokines. Sarcoma cells release tumour-derived EVs, facilitating communication, tumour progression, and inflammation. Methods: We conducted a literature search for keywords and concepts associated with EVs and inflammatory responses in sarcomas on the PubMed database. Keyword combinations included relevance to EVs, sarcomas and inflammation. Only studies from peer reviewed journals, written in English, and published between 2013-2024 were considered. Sarcoma subtypes viewed included: rhabdomyosarcoma, Ewing sarcoma, chondrosarcoma, liposarcoma, and osteosarcoma. Results: Ewing sarcoma EVs carry miRNA and mRNAs including EWS/FLI-1 mRNA, and surface proteins causing upregulation of inflammatory pathways, and contributing to the release of pro-inflammatory cytokines. Rhabdomyosarcoma EVs carry miRNAs and PAX3/7-FOXO1 mRNA stimulating inflammatory pathways to release cytokines and chemokines. Chondrosarcoma’s hypoxic TME causes increased EV secretion, favouring M2 polarisation, and the production of immunosuppressive markers. Liposarcoma EVs enriched with miRNAs and MDM2 induced an inflammatory response in macrophages. Osteosarcoma EVs contained TGF-β and miRNA cargo which influenced macrophage polarisation. Discussion: Sarcoma-derived EVs contain a wide range of mRNAs and miRNAs, with Ewing sarcoma and Rhabdomyosarcoma also containing the fusion oncogenes EWS/FLI-1 and PAX3/7-FOXO1, respectively. Surface proteins on Ewing sarcoma and osteosarcoma EVs were better understood compared to other sarcomas. Various sarcoma-derived EV cargo demonstrated potential to influence inflammatory pathways within macrophages. Macrophage polarisation is fundamental to immune function, determining pro-inflammatory and anti-inflammatory responses. Conclusion: By understanding the role of EVs in the inflammatory process, a deeper insight in tumour progression and tumour cell-cell communication can be achieved. The uniqueness of EV cargo to certain sarcoma subtypes could potentially indicate a future as biomarkers for early cancer detection. Further research into inflammatory effects by EVs can provide potential as novel therapeutics.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.004
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: none
GenreCandidate signal: Review · Consensus signal: Review
Teacher disagreement score0.013
Threshold uncertainty score0.015

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.004
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0020.002
Bibliometrics0.0130.011
Science and technology studies0.0010.001
Scholarly communication0.0020.002
Open science0.0010.001
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0050.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.040
GPT teacher head0.428
Teacher spread0.388 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designNot applicable
Domainnot available
GenreReview

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2024
Admission routes1
Has abstractyes

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