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Record W4404293436 · doi:10.1016/j.brain.2024.100103

Two for tau: Automated model discovery reveals two-stage tau aggregation dynamics in Alzheimer’s disease

2024· article· en· W4404293436 on OpenAlexfundno aff
Charles A. Stockman, Alain Goriely, Ellen Kuhl

Bibliographic record

VenueBrain Multiphysics · 2024
Typearticle
Languageen
FieldMedicine
TopicAlzheimer's disease research and treatments
Canadian institutionsnot available
FundersHORIZON EUROPE European Research CouncilNational Institute on AgingCanadian Institutes of Health ResearchNational Institute of Biomedical Imaging and BioengineeringEngineering Research CentersFoundation for the National Institutes of HealthNational Science FoundationNational Institutes of HealthAlzheimer's Disease Neuroimaging Initiative
KeywordsDiseaseNeuroscienceAlzheimer's diseaseDynamics (music)Computer sciencePsychologyMedicineInternal medicine

Abstract

fetched live from OpenAlex

Alzheimer’s disease is a neurodegenerative disorder characterized by the presence of amyloid- β plaques and the accumulation of misfolded tau proteins and neurofibrillary tangles in the brain. A thorough understanding of the local accumulation of tau is critical to develop effective therapeutic strategies. Tau pathology has traditionally been described using reaction–diffusion models, which succeed in capturing the global spread, but fail to accurately describe the local aggregation dynamics. Current mathematical models enforce a single-peak behavior in tau aggregation, which does not align well with clinical observations. Here we identify a more accurate description of tau aggregation that reflects the complex patterns observed in patients. We propose an innovative approach that uses constitutive neural networks to autonomously discover bell-shaped aggregation functions with multiple peaks from clinical positron emission tomography (PET) data of misfolded tau protein. Our method reveals previously overlooked two-stage aggregation dynamics by uncovering a two-term ordinary differential equation that links the local accumulation rate to the tau concentration. When trained on data from amyloid- β positive and negative subjects, the neural network clearly distinguishes between both groups and uncovers a more subtle relationship between amyloid- β and tau than previously postulated. In line with the amyloid–tau dual pathway hypothesis, our results show that the presence of toxic amyloid- β influences the accumulation of tau, particularly in the earlier disease stages. We expect that our approach to autonomously discover the accumulation dynamics of pathological proteins will improve simulations of tau dynamics in Alzheimer’s disease and provide new insights into disease progression. Significance Statement In Alzheimer’s disease, understanding the local dynamics of tau protein aggregation is crucial for developing effective treatments. Traditional models for tau protein dynamics use reaction–diffusion models that fail to accurately capture these local patterns. Our study introduces a novel approach that leverages constitutive neural networks to autonomously discover the complex, multi-peak aggregation dynamics from clinical PET data. This method reveals a previously overlooked two-stage tau accumulation process and a nuanced relationship between amyloid- β and tau. By distinguishing between amyloid- β positive and negative subjects, our model supports the amyloid–tau dual pathway hypothesis and offers novel insights into tau protein aggregation that have the potent to advance our understanding of Alzheimer’s disease progression.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Simulation or modeling · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.797
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.001
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.041
GPT teacher head0.370
Teacher spread0.329 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designSimulation or modeling
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations5
Published2024
Admission routes1
Has abstractyes

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