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Record W4407419055 · doi:10.1093/ijnp/pyae059.070

BEHAVIOURAL CONTROL TRAINING AND ENDOCANNABINOID ENHANCEMENT LEAD TO ANTIDEPRESSANT/ANXIOLYTIC-LIKE REVERSAL OF CHRONIC STRESS-INDUCED BEHAVIOURAL DEFICITS

2025· article· en· W4407419055 on OpenAlexaff
Tadhg Strand, Gavin Afonso, C. De Oliveria, Matheus Cavatti, José N. Nóbrega, Francis Rodriguez Bambico

Bibliographic record

VenueThe International Journal of Neuropsychopharmacology · 2025
Typearticle
Languageen
FieldPsychology
TopicMindfulness and Compassion Interventions
Canadian institutionsCentre for Addiction and Mental HealthMemorial University of Newfoundland
Fundersnot available
KeywordsEndocannabinoid systemAnxiolyticAntidepressantNeurosciencePsychologyMedicinePhysical medicine and rehabilitationPsychiatryAnxietyInternal medicineHippocampus

Abstract

fetched live from OpenAlex

Abstract Background A cognitive model of stress posits that psycho-affective disorders can arise from a perceived lack of control over stressors [1]. Stress uncontrollability is linked to perturbed prefrontocortical (PFC)-raphe signaling of endocannabinoids, e.g., anandamide [2]. Conversely, behavioural therapies such as cognitive-behavioural and remediation therapies ameliorate mood-related symptoms by enhancing perceived stress controllability [3]. There is also evidence that pharmacologically augmenting brain endocannabinoid levels results in antidepressant and anti-stress action [2],[3].Objectives: We developed a rodent model of cognitive remediation based on the yoked inescapable stress (IES)– escapable stress (ES) paradigm, and on a modified Morris water maze (with vs. without an escape platform). Methods: Animals were first exposed to 6 weeks of chronic unpredictable stress (CUS), then were submitted to behavioural control training (BCT), where BCT+ animals were repeatedly allowed to learn to evade the stressors, while BCT- were subjected to inescapable stressors. CUS-exposed animals exhibited depressive-like (sucrose preference, fruit loops and forced swim tests) and anxiety-like reactivity (novelty-suppressed feeding and elevated plus maze), and increased serum corticosterone levels. Some mice were subjected to in vivo glucose biosensor monitoring of the PFC in response to a challenge injection of the cannabinoid CB1 receptor antagonist, AM251 (1 mg/kg, ip). Results CUS-induced behavioral deficits were reversed by 8-10 days but not by 3 days of BCT in CUS- exposed BCT+ animals and not in CUS-exposed BCT- animals. In non-CUS animals, BCT+ and BCT- led to slight and transient behavioural deficits. We have determined that behavioural improvements in BCT+ were associated with a blunting of AM251-induced decrease in PFC glucose levels. We therefore assessed changes in the PFC by electrophysiological recordings of PFC and raphe neuronal activity after CUS and BCT (BCT+ vs. BCT-; data to follow). Lastly, we found that in CUS mice, a behaviorally inert dose of the anandamide-enhancing fatty acid amide hydrolase (FAAH) inhibitor, URB597 (0.05 mg/kg, ip) when combined with 3 days of BCT reversed the CUS deficits.Discussion: These data show that cognitive therapies and BCT can be modeled in rodents, and that these effects may be mediated by increased endocannabinoid signaling in the PFC, similar to those induced by the fast-acting antidepressant ketamine [4]. BCT and URB597 may therefore have synergistic therapeutic activity. Because there may be issues associated with the repeated use of ketamine in the clinic [5], this combinatory strategy could serve as a promising alternative.Conclusion: This study provides a preclinical framework with which to test whether cognitive therapies and endocannabinoid enhancing drugs could be combined to yield faster onset and more stable expression of the therapeutic response. References [1]Maier, S.F. (1984) 'Learned helplessness and animal models of depression’, Progress in Neuro- Psychopharmacology and Biological Psychiatry, 8(3), pp. 435–446. Available at: https://doi.org/10.1016/S0278-5846(84)80032-9. [2]Bambico, F.R. and Gobbi, G. (2008) 'The cannabinoid CB1 receptor and the endocannabinoid anandamide: possible antidepressant targets’, Expert Opinion on Therapeutic Targets, 12(11), pp. 1347–1366. Available at: https://doi.org/10.1517/14728222.12.11.1347. [3]Addis, M.E. and Carpenter, K.M. (2000) 'The treatment rationale in cognitive behavioral therapy: Psychological mechanisms and clinical guidelines’, Cognitive and Behavioral Practice, 7(2), pp. 147–156. Available at: https://doi.org/10.1016/S1077-7229(00)80025-5. [3]Gobbi, G. et al. (2005) 'Antidepressant-Like Activity and Modulation of Brain Monoaminergic Transmission by Blockade of Anandamide Hydrolysis’, Proceedings of the National Academy of Sciences of the United States of America, 102(51), pp. 18620–18625. [4]Browne, C. and Lucki, I. (2013) 'Antidepressant effects of ketamine: mechanisms underlying fast-acting novel antidepressants’, Frontiers in Pharmacology, 4. Available at: https://www.frontiersin.org/articles/10.3389/fphar.2013.00161 (Accessed: 14 October 2023). [5]Morgan, C.J.A., Curran, H.V. and Drugs (ISCD), the I.S.C. on (2012) 'Ketamine use: a review’, Addiction, 107(1), pp. 27–38. Available at: https://doi.org/10.1111/j.1360-0443.2011.03576.x.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.007

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.002
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.047
GPT teacher head0.368
Teacher spread0.321 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2025
Admission routes1
Has abstractyes

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