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Record W4408508497 · doi:10.1093/brain/awaf102

Large-scale profiling of antibody reactivity to glycolipids in patients with Guillain-Barré syndrome

2025· article· en· W4408508497 on OpenAlexaff
Robin C.M. Thomma, Susan K. Halstead, L.C. de Koning, Evelin E J A Wiegers, Dawn Gourlay, Anne P. Tio‐Gillen, Wouter van Rijs, Henning Andersen, Giovanni Antonini, Samuel Arends, Shahram Attarian, Fábio Barroso, Kathleen Bateman, Luana Benedetti, Peter Van den Bergh, Jan Bürmann, Mark Busby, Carlos Casasnovas, Efthimios Dardiotis, Amy Davidson, Thomas E. Feasby, Janev Fehmi, Giuliana Galassi, Tania García‐Sobrino, Volkan Granit, Gerardo Gutiérrez‐Gutiérrez, Robert D. M. Hadden, Thomas Harbo, Hans‐Peter Hartung, Imran Hasan, James Holt, Zhahirul Islam, Summer Karafiath, Hans Katzberg, Noah Kolb, Susumu Kusunoki, Satoshi Kuwabara, Motoi Kuwahara, Helmar C. Lehmann, Sonja E. Leonhard, L. Aguilar, Soledad Monges, Eduardo Nobile‐Orazio, Julio Pardo, Yann Péréon, Luís Querol, Ricardo Reisin, Simon Rinaldi, Paolo Ripellino, Rhys Roberts, Olivier Scheidegger, Nortina Shahrizaila, Kazim A. Sheikh, Nicholas J. Silvestri, Søren H. Sindrup, Beth Stein, Cheng‐Yin Tan, Hatice Tankişi, Leo H. Visser, Waqar Waheed, Ruth Huizinga, Hugh J. Willison, Jean Addington, Senda Ajroud‐Driss, Suzanne Arends, S. Attarian, Umesh A. Badrising, Claudia Balducci, Kevin P. Bateman, I.R. Bella, Benno van den Berg, T. E. Bertoríni, Ratna Bhavaraju‐Sanka, Federica Bozzano, Thomas H. Brannagan, Chiara Briani, J. Bürmann, S. Butterworth, Giovanna Capodivento, Carlos Casasnovas, Guido Cavaletti, Chi‐Chao Chao, Shiping Chen, Elisa Cisneros, Kristl G. Claeys, M E Conti, David R. Cornblath, Jeremy Cosgrove, M. C. Dalakas, Philip Van Damme, Andrew R. Davidson, Gert W. van Dijk, Mazen M. Dimachkie, Alex Y. Doets, P.A. van Doorn, Andoni Echaniz‐Laguna, Filip Eftimov, Catharina G. Faber, Rosa Fazio, J Fernández-Travieso, Chris Fokke, Tatsuya Fujioka, E. Fulgenzi, Marcel P.J. Garssen, Claudia Giannotta, C.J. Gijsbers, Jennifer Gilchrist, H Job Gilhuis, Janna Goldstein, Kenneth C. Gorson, Namita Goyal, Aude‐Marie Grapperon, G Guttiérrez-Guttiérrez, L. Gutman, Hans‐Peter Hartung, Shoma Hayat, Rudi W. Hendriks, Jakob Vormstrup Holbech, James K. L. Holt, Sung‐Tsang Hsieh, M. Htut, R. A. C. Hughes, Robert B. Huizinga, Andrea M. Humm, Thomas Hundsberger, Badrul Islam, Zahidul Islam, Bram Jacobs, Israt Jahan, Korné Jellema, I. Jericó Pascual, K Kaida, Henk Kerkhoff, Mohammad Khoshnoodi, Lynette Kiers, Norito Kokubun, Rinske van Koningsveld, Anneke J. van der Kooi, J.C.H.M. Kramers, Krista Kuitwaard, T Kuntzer, S Kusunoki, Jing Yi Kwan, Shafeeq Ladha, Lisbeth Lassen, Agustina M. Lascano, Victoria Lawson, C Lleixa-Rodriguez, Linda W.G. Luijten, Michael P. Lunn, Armelle Magot, Hadi Manji, Cintia Marchesoni, Girolama Alessandra Marfia, Celedonio Márquez‐Infante, Eugenia Martínez‐Hernández, G Mataluni, M. Mattiazi, Christopher McDermott, Gregg Meekins, James Miller, Quazi Deen Mohammad, M.S. Monges, Parameswaran Nair, Caterina Nascimbene, Lucilla Nobbio, R.J. Nowak, M. Osei-Bonsu, José Ramón de Juanes Pardo, Farah Pelouto, Melanie S. Pulley, Stephen Reddel, T. van der Ree, I. Rojas-Marcos, Joyce Roodbol, Stacy A. Rudnicki, Gabriele Sachs, Johnny P.A. Samijn, L. Santoro, A. Savransky, Angelo Schenone, Lenka Schwindling, María J. Sedano Tous, Vasileios Siokas, Claudia Sommer, Beth E. Shubin Stein, Amro Stino, Tomoki Suichi, P Tsouni, Paul Twydell, J.D. Varrato, J C Verboon, Camiel Verhamme, Frédérique H Vermeij, Jan J.G.M. Verschuuren, M.V. Vytopil, Waquas Waheed, Christa Walgaard, Yuzhuo Wang, Eveline Wiegers, H J Willison, Paul W. Wirtz, M. van Woerkom, Yuki Yamagishi, Keisuke Yoshikawa, L L Zhang, Lan Zhou, Slađana Živković

Bibliographic record

VenueBrain · 2025
Typearticle
Languageen
FieldMedicine
TopicPeripheral Neuropathies and Disorders
Canadian institutionsUniversity of TorontoUniversity Health NetworkUniversity of Calgary
FundersGrifolsFonds de dotation CSL Behring pour la rechercheAxencia Galega de InnovaciónCSL BehringUniversity of GlasgowErasmus Medisch CentrumGBS/CIDP Foundation InternationalWellcome Trust
KeywordsGlycolipidGuillain-Barre syndromeAntibodyGangliosideImmunologyAntigenMedicinePolyradiculoneuropathyBiologyBiochemistry

Abstract

fetched live from OpenAlex

Guillain-Barré syndrome is an acute polyradiculoneuropathy in which preceding infections often elicit the production of antibodies that target peripheral nerve antigens, principally gangliosides. Anti-ganglioside antibodies are thought to play a key role in the clinical diversity of the disease and can be helpful in clinical practice. Extensive research into clinical associations of individual anti-ganglioside antibody specificities has been performed. Recent research has highlighted glycolipid complexes, glycolipid combinations that may alter antibody binding, as targets. In this study, we investigated antibody reactivity patterns to glycolipids and glycolipid complexes using combinatorial array, in relation to clinical features in Guillain-Barré syndrome. In total, 1413 patients from the observational International Guillain-Barré syndrome Outcome Study (0-91 years, 60.3% male) and 1061 controls (healthy, family, infectious, vaccination, other neurological disease) were included. Acute-phase sera from patients were screened for IgM, IgG, and IgA reactivity against 15 glycolipids and one phospholipid and their heteromeric complexes, similarly to archived control sera. Antibody specificities and reactivity patterns were analysed in relation to clinical features. Of all patients, 1309 (92.6%) were positive for at least one anti-glycolipid (complex) antibody. Anti-GM1 and anti-GQ1b (complex) antibodies best distinguished motor Guillain-Barré syndrome and Miller Fisher syndrome from controls, with antibodies to glycolipid complexes outperforming antibodies to single glycolipids. Three models consisting of anti-glycolipid (complex) antibodies distinguished patients with Guillain-Barré syndrome, the motor variant, and Miller Fisher syndrome from controls with high sensitivity and specificity, performing better than antibodies to single glycolipids used in clinical practice. Seven patient clusters with particular antibody reactivity patterns were identified. These clusters were distinguished by geographical region, clinical variants, preceding Campylobacter jejuni infection, electrophysiological subtypes, the Medical Research Council sum score at study entry, and the ability to walk 10 m unaided at 26 weeks. Two patient clusters with distinct anti-GM1 (complex) reactivity (broad versus restricted) differed in frequency of the axonal subtype. In cumulative incidence analyses, 15 anti-glycolipid (complex) antibodies were associated with the time required to regain the ability to walk 10 m unaided. After adjustment for known prognostic factors, IgG anti-GQ1b:GM4, GQ1b:PS and GQ1b:Sulfatide remained associated with faster recovery. Addition of anti-glycolipid antibodies to clinical prognostic models slightly improved their discriminative capacity, though insufficiently to improve the models. Measurement of anti-glycolipid antibodies by combinatorial array increases the diagnostic yield compared to assaying single glycolipids, identifies clinically relevant antibody reactivity patterns to glycolipids and glycolipid complexes, and may be useful in outcome prediction in Guillain-Barré syndrome.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.002

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.001
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.005
GPT teacher head0.259
Teacher spread0.255 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations10
Published2025
Admission routes1
Has abstractyes

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