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Record W4409624323 · doi:10.1158/1538-7445.am2025-953

Abstract 953: Oncolytic viruses as in situ personalized tumor vaccines for durable efficacy

2025· article· en· W4409624323 on OpenAlexaff
William Jia

Bibliographic record

VenueCancer Research · 2025
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicVirus-based gene therapy research
Canadian institutionsVancouver Biotech (Canada)
Fundersnot available
KeywordsOncolytic virusMedicineVirologyCancer researchVirus

Abstract

fetched live from OpenAlex

Tumor vaccines have shown clinical promise in treating certain cancers. However, their long-term effectiveness is often hindered by two major challenges: 1. The immune-suppressive tumor microenvironment (TME). 2. Selective targeting of antigen-positive tumor cells, which can lead to the expansion of antigen-negative cells and eventual tumor relapse. To address these challenges, we explored a combination approach using mRNA tumor vaccines and oncolytic virotherapy in HPV and HER2 mouse tumor models. Our findings reveal that while mRNA vaccines effectively eradicate antigen-positive tumors and suppress tumor growth, resistance quickly emerges, enabling tumors to evade the vaccine's effects. When mRNA vaccination was followed by intratumoral administration of an oncolytic virus—either with or without HPV or HER2 antigen expression—a significantly more durable anti-tumor response was achieved in both models. The oncolytic virus initiated broad tumor antigen presentation by lysing tumor cells, releasing diverse tumor-associated antigens, and increasing the overall antigenic load within the tumor. Furthermore, the lytic activity of the virus reprogrammed the TME, transforming it from an immune-suppressive environment into one more favorable for anti-tumor immune responses. This combination therapy elicited a potent local immune response, leveraging enhanced antigen presentation by the virus. The modified TME allowed the systemic immune response generated by the mRNA vaccine to penetrate and act effectively within the tumor. Additionally, the treatment induced systemic antigen-specific T-cell responses, and treated animals developed resistance to subsequent tumor challenges, demonstrating the establishment of immune memory. Our study highlights the unique role of oncolytic virotherapy as an intratumoral "boost" mechanism. By lysing tumor cells, oncolytic viruses amplify the range of tumor antigens presented to the immune system and reshape the TME to support robust immune activity. This synergy between mRNA vaccination and oncolytic virotherapy—when employed in a prime-boost regimen—produces a powerful and durable anti-tumor immune response that is both localized and systemically protective against recurrence. In conclusion, this research underscores the potential of combining mRNA tumor vaccines with oncolytic virotherapy to overcome immune evasion mechanisms, enhance tumor antigen presentation, and reprogram the TME, providing a promising strategy for achieving long-lasting anti-tumor immunity. Citation Format: William WEI GUO Jia. Oncolytic viruses as in situ personalized tumor vaccines for durable efficacy [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2025; Part 1 (Regular Abstracts); 2025 Apr 25-30; Chicago, IL. Philadelphia (PA): AACR; Cancer Res 2025;85(8_Suppl_1):Abstract nr 953.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: none
GenreCandidate signal: Other · Consensus signal: none
Teacher disagreement score0.003
Threshold uncertainty score0.011

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0030.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.068
GPT teacher head0.451
Teacher spread0.383 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designNot applicable
Domainnot available
GenreOther

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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