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Record W4409690397 · doi:10.1158/1538-7445.am2025-299

Abstract 299: Characterization of a senescence phenotype induced by advanced glycation end products in prostate cancer

2025· article· en· W4409690397 on OpenAlexaff
Luisa F. Guzman Bacca, Tarek Hallal, Carole Luthold, Nadia Boufaied, François Bordeleau, David P. Labbé

Bibliographic record

VenueCancer Research · 2025
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicAdvanced Glycation End Products research
Canadian institutionsMcGill University Health CentreUniversité LavalMcGill University
Fundersnot available
KeywordsGlycationProstate cancerSenescenceCancerCancer researchPhenotypeMedicineBiologyInternal medicineBiochemistryGeneReceptor

Abstract

fetched live from OpenAlex

Prostate cancer (PCa) represents a worldwide leading cause of cancer-related mortality in men. Diabetes is also a highly prevalent and a rapidly growing pathology. Intriguingly, diabetes is associated with decreasing PCa incidence, and at the same time, it is linked to increased mortality rates for men already afflicted by the disease. A plausible mediator causing this effect are Advanced Glycation End Products (AGEs), products of the Maillard reaction, occurring between sugars and proteins at body temperature. AGEs, known to be elevated in diabetic patients, have been reported to increase the Extracellular Matrix (ECM) stiffness via ECM-crosslinking, potentially explaining the tumor-promoting effects of diabetes in cancer progression. Nonetheless, the protective role of diabetes in prostate cancer is not yet fully understood. Interestingly, we demonstrated that AGEs, produced by mixing sugars and an abundant circulating protein in the blood decreased PCa cell proliferation in vitro in a dose-dependent and irreversible manner across a panel of human PCa cell lines. Importantly, through transcriptomic analysis, we found that AGEs-mediated cell proliferation blocking correlate with a senescence transcriptional signature. Accordingly, treatment with AGEs lead to increased beta-galactosidase activity, a hallmark of senescence, and to the upregulation of senescent markers linked to cell cycle arrest (p21WAF1/Cip1) and pro-inflammatory cytokines (IL-8) at the transcriptional and translational levels. We also confirmed the expression of the senescence-associated secretory phenotype (SASP, e.g., IL, CXCL, CCL) in AGEs-treated PCa cells. Finally, our validation of key transcriptomics analysis confirmed the upregulation of HMOX1, an enzyme reported to be induced during cellular senescence, upon AGEs treatment. Altogether, our results suggest that AGEs-mediated senescence could underlie the lower incidence of PCa among diabetic patients. This sets the stage for combination treatments between AGEs and senolytics, drugs that selectively target and induce cell death in senescent cells, as a novel therapeutic strategy in PCa. Citation Format: Luisa Bacca, Tarek Hallal, Carole Luthold, Nadia Boufaied, François Bordeleau, David Labbé. Characterization of a senescence phenotype induced by advanced glycation end products in prostate cancer [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2025; Part 1 (Regular Abstracts); 2025 Apr 25-30; Chicago, IL. Philadelphia (PA): AACR; Cancer Res 2025;85(8_Suppl_1):Abstract nr 299.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.001
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.066
Threshold uncertainty score0.568

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.001
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.032
GPT teacher head0.385
Teacher spread0.354 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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