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Abstract LB019: LINC00152-regulated PDE4D mediates drug resistance and metastasis in highly aggressive breast cancer

2025· article· en· W4409821538 on OpenAlexaff
Özge Saatci, Rashedul Alam, Kim‐Tuyen Huynh‐Dam, Aynur Işık, Meral Üner, Nevin Belder, Pelin G. Ersan, Ünal Metin Tokat, Bürge Ulukan, Metin Çetįn, Kübra Çalışır, Mustafa Emre Gedik, Hilal Bal, Ozlem Sener Sahin, Yasser Riazalhosseini, Denis Thieffry, Daniel Gautheret, Besim Öğretmen, Sercan Aksoy, Ayşegül Üner, Aytekin Akyol, Özgür Şahin

Bibliographic record

VenueCancer Research · 2025
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicPhosphodiesterase function and regulation
Canadian institutionsConcordia UniversityMcGill University
Fundersnot available
KeywordsMedicineBreast cancerMetastasisCancerOncologyBreast cancer metastasisDrug resistanceInternal medicineCancer researchPharmacologyCancer metastasisBiologyMicrobiology

Abstract

fetched live from OpenAlex

Abstract Phosphodiesterase 4D (PDE4D) is a member of the phosphodiesterase family of enzymes, catalyzing the hydrolysis of cAMP second messenger and inhibiting cAMP signaling. Targeting PDE4D raises the intracellular cAMP levels, leading to apoptosis and cell cycle arrest in different tumor types. However, its contribution to drug resistance and metastasis is still elusive. lncRNAs are more than 200 nucleotides in length and carry out diverse functions including transcriptional regulation, and regulation of proteins or RNA molecules by direct binding and stabilization. LINC00152 is an oncogenic lncRNA that promotes survival, proliferation, epithelial-mesenchymal transition (EMT) and invasiveness in cancer cells. Despite being a driver in several key oncogenic processes, whether LINC00152 is an upstream regulator of PDE4D to drive endocrine resistance and metastasis in ER+ breast cancer remains to be elucidated. Here we showed that LINC00152 stabilizes PDE4D mRNA, thus driving tamoxifen resistance upon deactivation of the cAMP/PKA/CREB axis. Overexpressing PDE4D rescues LINC00152-mediated tamoxifen resistance by blocking tamoxifen-induced ferroptosis. In addition, we demonstrated that inhibiting LINC00152 or PDE4D reduces the migration of endocrine resistant cells upon PKA-mediated blockage of TGF-β signaling. Targeting PDE4D using the clinically-tested PDE4D selective inhibitor, BPN14770 significantly reduces spontaneous metastasis in the highly aggressive, endocrine resistant-mimicking MMTV-PyMT model in vivo. Importantly, we showed that high levels of PDE4D mRNA is associated with worse metastasis-free survival in endocrine-treated ER+ breast cancer patients. Overall, we identified the highly oncogenic lncRNA, LINC00152 as an upstream regulator of PDE4D, which drives endocrine resistance and metastasis in the highly aggressive ER+ breast cancer. Citation Format: Ozge Saatci, Rashedul Alam, Kim-Tuyen Huynh-Dam, Aynur Isik, Meral Uner, Nevin Belder, Pelin Ersan, Unal M. Tokat, Burge Ulukan, Metin Cetin, Kubra Calisir, Mustafa E. Gedik, Hilal Bal, Ozlem Sener Sahin, Yasser Riazalhosseini, Denis Thieffry, Daniel Gautheret, Besim Ogretmen, Sercan Aksoy, Aysegul Uner, Aytekin Akyol, Ozgur Sahin. LINC00152-regulated PDE4D mediates drug resistance and metastasis in highly aggressive breast cancer [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2025; Part 2 (Late-Breaking, Clinical Trial, and Invited Abstracts); 2025 Apr 25-30; Chicago, IL. Philadelphia (PA): AACR; Cancer Res 2025;85(8_Suppl_2):Abstract nr LB019.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.320
Threshold uncertainty score0.522

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.018
GPT teacher head0.350
Teacher spread0.332 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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