MétaCan
Menu
← Back to cohort
Record W4410277377 · doi:10.1101/2025.05.05.25326880

Unravelling the molecular mechanisms causal to type 2 diabetes across global populations and disease-relevant tissues

2025· preprint· en· W4410277377 on OpenAlexafffund
Ozvan Bocher, Ana Luiza Arruda, Satoshi Yoshiji, Chi Zhao, Chen‐Yang Su, Xianyong Yin, Davis Cammann, Henry J. Taylor, Jingchun Chen, K. Suzuki, Ravi Mandla, Alicia Huerta‐Chagoya, Ta‐Yu Yang, Fumihiko Matsuda, Josep M. Mercader, Jason Flannick, James B. Meigs, Alexis C. Wood, Marijana Vujković, Benjamin F. Voight, Cassandra N. Spracklen, Andrew P. Morris, Eleftheria Zeggini

Bibliographic record

VenuemedRxiv · 2025
Typepreprint
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicGenetic Associations and Epidemiology
Canadian institutionsMcGill UniversityMcGill Genome Centre
FundersNational Center for Advancing Translational SciencesNational Heart, Lung, and Blood InstituteNational Institute for Health and Care ResearchHelmholtz Zentrum MünchenJapan Society for the Promotion of ScienceManchester Biomedical Research CentreEuropean CommissionAmerican Diabetes AssociationCanadian Institutes of Health ResearchNational Institutes of HealthTD BankVersus ArthritisMedical Research Council
KeywordsDiseaseType 2 diabetesComputational biologyType (biology)Evolutionary biologyDiabetes mellitusBiologyMedicineInternal medicineEcologyEndocrinology

Abstract

fetched live from OpenAlex

Type 2 diabetes (T2D) is a prevalent disease that arises from complex molecular mechanisms. Here, we leverage T2D multi-ancestry genetic associations to identify causal molecular mechanisms in an ancestry- and tissue-aware manner. Using two-sample Mendelian Randomization corroborated by colocalization across four global ancestries, we analyze 20,307 gene and 1,630 protein expression levels using blood-derived cis -quantitative trait loci (QTLs). We detect causal effects of genetically predicted levels of 335 genes and 46 proteins on T2D risk, with 16.4% and 50% replication in independent cohorts, respectively. Using gene expression cis-QTLs derived from seven T2D-relevant tissues, we identify causal links between the expression of 676 genes and T2D risk, including novel associations such as CPXM1 , PTGES2 and FAM20B . Causal effects are mostly shared across ancestries, but highly heterogeneous across tissues. Our findings provide insights in cross-ancestry and tissue-informed multi-omics causal inference analysis approaches and demonstrate their power in uncovering molecular processes driving T2D.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.004
metaresearch head score (Gemma)0.007
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.004
Threshold uncertainty score0.019

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0040.007
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0010.002
Science and technology studies0.0010.001
Scholarly communication0.0010.001
Open science0.0000.002
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0030.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.022
GPT teacher head0.321
Teacher spread0.299 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2025
Admission routes2
Has abstractyes

Explore more

Same venuemedRxiv→Same topicGenetic Associations and Epidemiology→French-language works237,207→