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Record W4410715729 · doi:10.3899/jrheum.2025-0390.o047

NOVEL AUTOANTIBODIES IDENTIFIED IN THE ANTIPHOSPHOLIPID SYNDROME

2025· article· en· W4410715729 on OpenAlexvenueno aff
Shikai Hu, Yangzhong Zhou, Menghua Cai, Jiuliang Zhao

Bibliographic record

VenueThe Journal of Rheumatology · 2025
Typearticle
Languageen
FieldMedicine
TopicSystemic Lupus Erythematosus Research
Canadian institutionsnot available
Fundersnot available
KeywordsMedicineAutoantibodyAntiphospholipid syndromeImmunopathologyImmunologyAutoimmune diseaseConnective tissue diseaseAntibody

Abstract

fetched live from OpenAlex

O047 / #343 Topic:AS03 - Antiphospholipid Syndrome ABSTRACT CONCURRENT SESSION 08: RECENT ADVANCES IN LUPUS BIOMARKERS 23-05-2025 1:40 PM - 2:40 PM Background/Purpose The antiphospholipid syndrome (APS) is a systemic autoimmune disease characterized by arterial, venous, or microvascular thrombosis, recurrent pregnancy morbidity, or nonthrombotic manifestations in the setting of persistent antiphospholipid antibodies (aPL), namely anti-β2 glycoprotein-I antibody (aβ2GPI), anticardiolipin antibody (aCL), and lupus anticoagulant (LAC). Around one-third of the APS patients had an isolated LAC positivity lacking aβ2GPI and aCL. This study aimed to identify novel autoantibodies in APS using protein microarray technology. Methods Sera from APS patients, disease controls (DCs), and healthy controls (HCs) were applied to the HuProt™ Human Proteome Microarray v4.0 for the discovery of novel autoantibodies. Candidate autoantibodies were then validated using ELISA in an additional 372 sera samples, comprising 189 primary APS patients, 87 secondary APS patients, 48 SLE patients, and 48 HCs. Results During the discovery phase, HuProt microarrays were incubated with serum samples (5:1 mixture) from APS patients, DCs, and HCs to identify APS-associated autoantigens. Approximately 20 autoantigens were identified for subsequent validation. These potential autoantigens include proteins involved in or associated with ubiquitination and deubiquitination (UBE3A, ATXN3), proteasome (PSME3), microtubule (MAP9), vesicular transport (ASAP2), ribosome (NPM1, RPLP2), amino acid modification (PRMT7), DNA and RNA (RBM38, IRX2, AGO1), inflammation (WDR54, IRAK4, ACVR2B, N4BP1, MX1), metabolism (ACSBG1, SULT2B1, HK1, GLOD4), coagulation factor (SERPINB2), and others. In the validation phase, 6 proteins (SULT2B1, NPM1, AGO1, SERPINB2, ACVR2B, IRAK4) were selected and validated using ELISA. Anti-SULT2B1 and anti-AGO1 autoantibodies were significantly higher in primary APS patients. Anti-SULT2B1, anti-AGO1, and anti-NPM1 were also significantly higher in secondary APS patients. Anti-NPM1 positive patients exhibited a significantly higher incidence of SLE (50.9% vs 33.7%, p=0.013), cardiac valve involvement (16.3% vs 6.1%, p=0.031), and triple positivity (53.1% vs 33.5%, p=0.010) compared to anti-NPM1 negative patients. Validation of other autoantibodies is ongoing (Figure). Figure. Conclusions We identified novel autoantibodies targeting proteins involved in a broad range of biological processes in APS. Anti-SULT2B1, anti-AGO1, and anti-NPM1 autoantibodies were identified in APS patients, demonstrating diagnostic and clinical value. Further validation of additional autoantibodies in larger APS cohorts is ongoing.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.005

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.001
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.023
GPT teacher head0.321
Teacher spread0.297 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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