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The effects of age and exercise training on lysosomal adaptations within skeletal muscle

2025· article· en· W4411543772 on OpenAlexaffabout
Anastasiya Kuznyetsova, Thulasi Mahendran, David A. Hood

Bibliographic record

VenuePhysiology · 2025
Typearticle
Languageen
FieldPsychology
TopicChildren's Physical and Motor Development
Canadian institutionsYork University
Fundersnot available
KeywordsSkeletal muscleTraining (meteorology)Physical medicine and rehabilitationBiologyEndocrinologyPsychologyPhysiologyNeuroscienceMedicineInternal medicine

Abstract

fetched live from OpenAlex

Skeletal muscle mass comprises a large fraction (~40%) of the total human body mass, making it an essential contributor to overall physical health. Mitochondria are responsible for the metabolic status of skeletal muscle due to their ability to adapt to various physiological conditions. The aging population undergoes a loss of skeletal muscle mass, strength, and function, termed sarcopenia. The maintenance of a healthy mitochondrial pool is imperative for the preservation of muscle mass and metabolic health with age. The important removal of dysfunctional mitochondria happens via the process of mitophagy in which these organelles are tagged for degradation, engulfed and delivered to lysosomes, representing the vital role lysosomes play in the turnover of mitochondria. If lysosomes become defective with age, this can contribute to the accumulation of defective mitochondria. Thus, we have investigated lysosomes to understand their role in aging and exercise. We hypothesize that lysosome content will increase with age, but that lysosome proteolytic function will be diminished, and that exercise training will serve to restore this functionality in aged muscle. We studied these adaptations in skeletal muscle from young (5-7 months) and aged (21-23 months) mice following a 6-week voluntary wheel running (VWR) protocol compared to an untrained group. During the training protocol, the aged group ran significantly less than the young group. Despite this, training attenuated the aging-induced loss of muscle mass, especially in the predominantly slow-twitch soleus muscle. During an acute exhaustive bout of exercise, young and old trained groups ran significantly more time than untrained groups, indicating the effectiveness of the training to induce adaptative changes. Lysosomal protein yield was 2-fold greater in the aged, compared to the young cohorts, and this was accompanied by 1.5-fold, 2-fold and 2-fold increases in Lamp1, mature cathepsin D and precursor cathepsin B in aged muscle respectively. Lysosome proteolytic function, assessed in purified lysosomal fractions, was reduced by ~30 % in aged muscle. Remarkably, training improved lysosomal function in both young (1.3- fold) and old (1.5-fold) muscle. Thus, our results showed that aged mice have increased lysosomal protein content while displaying a more dysfunctional organelle pool. The chronic exercise protocol induced mitigation of these changes, illustrating the potential of improved clearance of dysfunctional cargo, including mitochondria, via enhanced proteolytic capacity of lysosomes, thereby improving muscle health with age. Supported by NSERC Canada. This abstract was presented at the American Physiology Summit 2025 and is only available in HTML format. There is no downloadable file or PDF version. The Physiology editorial board was not involved in the peer review process.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.975
Threshold uncertainty score0.268

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.014
GPT teacher head0.271
Teacher spread0.257 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes2
Has abstractyes

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