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Record W4411846639 · doi:10.3899/jrheum.2025-0314.83

A Case of Familial Cold Autoinflammatory Syndrome Type 2

2025· article· en· W4411846639 on OpenAlexaffvenue
Michael Schinold, Sarah Oberholtzer

Bibliographic record

VenueThe Journal of Rheumatology · 2025
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicInflammasome and immune disorders
Canadian institutionsUniversity of Saskatchewan
Fundersnot available
KeywordsMedicineDermatology

Abstract

fetched live from OpenAlex

Background Systemic rheumatic diseases exist along a spectrum of autoimmune and autoinflammatory disorders. Autoinflammatory diseases (AID) result from errors of the innate immune system, which results in systemic inflammation. This differs from autoimmune syndromes which result from errors of the adaptive immune system when self-tolerance is broken and autoantibodies are generated. Case Mrs. X, a 46yo F, was referred for further evaluation and treatment of a possible cold autoinflammatory syndrome. She has a history of cold-induced episodes starting in her late childhood, which have worsened in severity since a SARS-CoV-2 infection in 2022. Her cold-induced episodes are marked by widespread hives and flushing, with associated arthralgias and myalgias, chest pain with concurrent tachycardia, and abdominal cramping with diarrhea. These episodes are self-limiting over the course of a day. She additionally has ongoing cyclic fevers, fatigue and headaches. Her physical examinations have always been unremarkable apart from these cold-induced episodes where she has widespread flushing of her skin and hives. Prior to this referral she has been evaluated by a number of other subspecialists whose investigations only revealed elevated serum amyloid A levels. She had genetic testing done looking for monogenic periodic fever syndromes, which revealed mutations of unknown significance in her NLRP12 gene, an area also mutated in her father’s testing. She has an extensive history of similar cold-induced episodes in other family members, which vary in severity, and follow an autosomal dominant pattern through her father’s family. No one in her family has received a formal diagnosis at this time. With this information, medical genetics agreed that this likely represents a cold autoinflammatory syndrome, familial cold autoinflammatory type 2, which is likely related to her NLRP12 mutation. Mrs. X has been using intermittent prednisone to treat these episodes with good effect previously. She was initiated on anakinra, an IL-1 receptor antagonist, which was effective at reducing the frequency and duration of her episodes, where she no longer uses prednisone. Conclusion Cold autoinflammatory syndromes have been described in literature, most often in pediatrics, with increasing frequency over the last decade. They are increasingly being studied which is revealing of genetic mutations as drivers of these diseases. This case highlights that adult presentations of these diseases exist and that the adult rheumatologist should have a working knowledge of these conditions and the genetics behind autoinflammatory disorders.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.002
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Case report · Consensus signal: Case report
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.005
Threshold uncertainty score0.017

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.002
Meta-epidemiology (narrow)0.0020.001
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0020.002
Science and technology studies0.0030.001
Scholarly communication0.0010.001
Open science0.0010.001
Research integrity0.0030.002
Insufficient payload (model declined to judge)0.0050.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.005
GPT teacher head0.233
Teacher spread0.227 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designCase report
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes2
Has abstractyes

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