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Record W4413284510 · doi:10.1093/ijnp/pyaf052.105

688. TEMPORAL IMMUNE EFFECTS OF ORAL KETAMINE ON PTSD: TRANSCRIPTOMIC EVIDENCE OF SHORT-TERM INFLAMMATION SUPPRESSION AND LONG-TERM IMMUNE REMODELLING

2025· article· en· W4413284510 on OpenAlexaff
Nathan J Wellington, Bonnie L. Quigley, Ana Paula Bouças, Jim Lagopoulos, Anna Kuballa

Bibliographic record

VenueThe International Journal of Neuropsychopharmacology · 2025
Typearticle
Languageen
FieldNeuroscience
TopicTryptophan and brain disorders
Canadian institutionsSt. Thomas Hospital
Fundersnot available
KeywordsTerm (time)InflammationImmune systemKetamineNeuroscienceMedicineImmunologyPsychology

Abstract

fetched live from OpenAlex

Abstract Background Ketamine has emerged as a promising rapid-acting treatment for post-traumatic stress disorder (PTSD), offering swift symptom relief. However, the molecular mechanisms underpinning its long-term therapeutic effects remain poorly understood. Aims & Objectives This study aimed to investigate the short- and long-term gene expression changes following oral ketamine treatment in individuals with PTSD. It was hypothesised that ketamine would induce time-dependent transcriptional changes, shifting from acute immune suppression to long-term immune modulation and tissue repair. Method Twenty-three individuals with PTSD (9 males, 14 females) participated in a six-week trial involving subanesthetic doses of oral ketamine. Peripheral blood mononuclear cells (PBMCs) were collected at baseline, one week (short-term), and four weeks (long-term) post-treatment. RNA sequencing and transcriptome-wide analysis were conducted to assess differential gene expression and pathway activity across timepoints. Results Analysis identified 533 up-regulated and 621 down-regulated genes across timepoints. A 37% increase in gene expression changes was observed from short- to long-term, along with a 6.5-fold increase in expression magnitude and an 8.8-fold rise in pathway activation. Key modulated pathways included interferon alpha/beta signalling (z = 4), IL-17 signalling (z = 3.36), cytokine storm (z = 4.26), neutrophil degranulation (z = 6.0), and antimicrobial peptide signalling (z = 1.63). Notable gene-level regulators included IL-6, IL-1β, IFI27, CXCL8, SOCS1/3, IL-10, and CAMP. Discussion & Conclusions Findings demonstrate that ketamine induces distinct temporal molecular changes, transitioning from short-term inflammation suppression and antimicrobial responses to long-term immune remodulation and tissue repair. These results provide critical insight into the prolonged therapeutic mechanisms of ketamine in PTSD and support the development of personalised maintenance strategies to sustain clinical benefit and prevent relapse.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.008

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.030
GPT teacher head0.341
Teacher spread0.311 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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