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Record W4414181287 · doi:10.1192/j.eurpsy.2025.1533

Shared Genetics between Depression and Cardiometabolic Disorders

2025· article· en· W4414181287 on OpenAlexaff
P. Kundi, Moin Syed, Bakhtiyar Alam Syed, Daljit Singh Sahota

Bibliographic record

VenueEuropean Psychiatry · 2025
Typearticle
Languageen
FieldMedicine
TopicBirth, Development, and Health
Canadian institutionsMcMaster UniversityPeace Arch HospitalVancouver Coastal Health
Fundersnot available
KeywordsDepression (economics)DiseaseObesityCoronary artery diseaseDiabetes mellitusType 2 diabetesMajor depressive disorderMood disordersBlood pressure

Abstract

fetched live from OpenAlex

Introduction Several studies have discovered associations between depression and cardiovascular disease risk factors and patients with Coronary Artery Disease with comorbid Depression have worse prognosis. There is growing evidence of polygenic overlap between depression, Coronary Artery Disease and other Cardiovascular risk factors and may suggest molecular mechanisms underlying the association between depression and raised cardiovascular disease risk. Objectives Depression and cardiometabolic disorders are both heritable and both are caused by a mix of genetic and environmental factors. Genetic factors contribute to 31–42 % in MDD (Sullivan et al. 2000 ), 30–60 % in coronary artery diseases (Marenberg et al. 1994 ), 26–69 % in type 2 diabetes (Almgren et al. 2011 ; Poulsen et al. 1999 ), 24–37 % in blood pressure (hypertension) (Van Rijn et al. 2007 ), 35–48 % in heart rate variability (Kupper et al. 2004 ), 40–70 % in obesity (body mass index) (Willyard 2014 ), and 58–66 % in the level of serum lipids (Knoblauch et al. 1997 ). Moreover, there are fairly high genetic co-heritabilities (genetic correlations) between depression and the different cardiometabolic disorders suggesting the influence of pleiotropic genes and shared biological pathways within them. Methods Literature search was conducted and appropriate material was then extracted to examine the hypothesis. Results Identification of shared molecular pathways supports a growing evidence base for cross-diagnostic treatment. Besides, further exploration of overlapping molecular pathophysiology can unveil novel targets for drug development and repurposing of existing medications. Also, cardiometabolic disorders can increase the risk of poor response to standard treatments in mood disorders. Lastly, studying shared pathways of depression and somatic disorders can untangle the clinical and genetic heterogeneity that underlies in these illnesses. Conclusions Genetic studies have suggested the involvement of pleiotropic genes in the comorbidity between depression and cardiometabolic disorders. While our abstract attempts to provide some insight into the common mechanisms and role of pleiotropic genes, in-depth understanding of how these genes mediate the association between depression and cardiometabolic diseases requires future larger scale comprehensive cross-disorder research. This will enable us to better understand why patients suffer from multiple diseases at a time and how multi-morbidities influence pharmacological treatment response to diseases. Disclosure of Interest None Declared

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.002
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.007
Threshold uncertainty score0.024

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.002
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0020.003
Science and technology studies0.0000.001
Scholarly communication0.0010.000
Open science0.0000.001
Research integrity0.0010.000
Insufficient payload (model declined to judge)0.0070.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.014
GPT teacher head0.289
Teacher spread0.276 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2025
Admission routes1
Has abstractyes

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