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Record W4414392126 · doi:10.1093/ejendo/lvaf168.091

P36 - Adavosertib-mediated wee1 inhibition as a strategy to overcome cisplatin resistance in non-seminoma testicular cancer: insights from preclinical models

2025· article· en· W4414392126 on OpenAlexaff
Mariangela Tamburello, C Depetris de Boldini, Andrea Abate, Valentina Salvi, Francesca Valcamonico, Maria Marcella Laganà, Deborah Cosentini, Nazareno Suardi, Giovanni Mirabella, Barbara Altieri, Sandra Sigala

Bibliographic record

VenueEuropean Journal of Endocrinology · 2025
Typearticle
Languageen
FieldMedicine
TopicTesticular diseases and treatments
Canadian institutionsSurgical Specialties (Canada)
Fundersnot available
KeywordsCisplatinWee1DNA damageCyclin-dependent kinase 1Cell cycleMitosisCell cycle checkpointViability assayCell

Abstract

fetched live from OpenAlex

Abstract Background/Introduction Testicular germ cell tumors (TGCTs) are the most common solid malignancy in young adult males with non-seminomatous representing a clinically aggressive subtype. Although cisplatin (CP)-based chemotherapy is highly effective, with cure rates exceeding 95%, a subset of patients develop resistance. The identification of therapeutic strategies to overcome CP-resistance remains an urgent clinical need. Purpose In this study, we evaluated the expression and pharmacological targeting of WEE1, a key regulator of the G2/M checkpoint and replication stress response, in non- seminoma cell models using the WEE1 inhibitor adavosertib (AZD1775). Methods Two non-seminoma cell lines, NCCIT and NT2/D1, and their CP-resistant subclones (NCCIT-R and NT2/D1-R) were used. The effects of adavosertib and CP, alone or in combination, on cell viability, cell-cycle progression, apoptosis, and DNA damage markers was assessed. Results WEE1 was expressed in non-seminoma cell models. Adavosertib reduced cell viability in a dose-dependent manner across all cell models in both 2D and 3D cultures. IC50 values were consistently in the low micromolar range with minimal variation (NCCIT: 0.550 μM; NCCIT-R: 0.630 μM; NT2/D1: 0.415 μM; NT2/D1-R: 0.630 μM). Adavosertib altered cell-cycle distribution, increasing S-phase population. Western blot analysis showed reduced CDK1 phosphorylation, indicating elevated CDK1 activity, with increased H3 phosphorylation, a mitotic marker. These findings suggest WEE1 inhibition disrupts cell cycle checkpoints, promotes replication stress, and drives cells with unrepaired DNA damage into premature mitosis, resulting in mitotic catastrophe. Caspase 3/7 activity was also increased after treatment, specifically at 24 hours, together with elevated markers of DNA damage (pH2Ax) and apoptosis (cleaved-PARP and cleaved-Caspase 3). In combination treatments, adavosertib enhanced CP efficacy across all models, including resistant lines. Notably, in NCCIT-R cells, CP IC50 dropped from 11.65 to 1.99 μM when combined with adavosertib, indicating a sixfold increase in CP-sensitivity. Conclusion(s) Overall, our findings demonstrate that WEE1 is expressed in non- seminoma models and is targetable. Pharmacological inhibition of WEE1 by adavosertib impairs cell viability, disrupts cell-cycle control, induces apoptosis, and restores CP-sensitivity in resistant models. These results provide compelling preclinical evidence supporting the combination of WEE1 inhibition with standard chemotherapy as a promising strategy to overcome CP-resistance in non-seminoma testicular cancer.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.495
Threshold uncertainty score0.754

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.026
GPT teacher head0.315
Teacher spread0.289 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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