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Record W4417019141 · doi:10.1182/blood-2025-6808

Inhibition of telomerase and ALT pathways in MDS/AML cell lines

2025· article· en· W4417019141 on OpenAlexaff
H Sow, Macoura Gadji, Brett L. Houston, Kaoru Tohyama, Sabine Mai

Bibliographic record

VenueBlood · 2025
Typearticle
Languageen
FieldMedicine
TopicTelomeres, Telomerase, and Senescence
Canadian institutionsUniversity of Manitoba
Fundersnot available
KeywordsTelomeraseTelomereCancer cellViability assayCell growthCell cultureProgrammed cell deathCell

Abstract

fetched live from OpenAlex

Abstract Background: The ability of human cancer cells to maintain telomere length over successive cell divisions rapidly emerged as an adaptive mechanism acquired by cancer cells to defy cell senescence and mortality. This mechanism of immortality of cancerous cells was first demonstrated in the 1990s. Telomerase activation is the most common pathway to avoid telomere length erosion and is used by 85% of all tumor cells, while alternative lengthening of telomere ALT is used by the remainder. Some tumors, albeit rare, demonstrate the co-activation of both telomere maintenance pathways. The consequence of continued cell proliferation of tumor cells despite their critically short telomeres allows for ongoing genomic instability and its propagation to the next generation of cells. We hypothesized that a consecutive inhibition of the two telomere maintenance pathways by first inhibiting telomerase followed by the ALT pathway inhibition will kill cancer cells. Methods: We performed successive inhibition of telomerase over time (0h-144h) followed by ALT pathway (72h-144h) on MDS-92, MDS-L (Myelodysplastic Syndromes), and OCI-AML3 (Acute Myeloid Leukemia) cell lines, and used Normal Human Primary Fibroblasts (GL51/92 Roe) as controls. Cell death was assayed by live cell imaging using NucGreen™ Dead 488 and trypan blue exclusion. Results: Consecutive inhibition of both telomere maintenance pathways showed a significant decrease in cell viability rate over 72h of treatment with 200 µM SB1532 that were 25% for MDS-L and 27% for MDS92 and 38% for OCI-AML3. Cell death was increased by adding 4 nM Trabectedin (ALT inhibition) for an additional 3 days, leading to the viability rate results at 144 hours that were 1% for MDS-L, 0% for MDS92, and 9% for OCI-AML3. Meanwhile, primary fibroblast cells at low and high passages did not show sensitivity to these molecules with a cell viability rate of 93% and 87% after 144 hours, respectively. Conclusion: Inhibition of both telomere maintenance pathways in MDS/AML cell lines demonstrated a decrease in cell viability and increased cell death while primary fibroblasts showed no significant effect on survival. In future, study of primary cells from patients is necessary to confirm these results and to pave the way for cancer treatment in hematological and solid tumor neoplasias with molecules and/or small molecules targeting telomerase and ALT pathways.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.005

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.012
GPT teacher head0.238
Teacher spread0.226 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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