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Record W6921983573 · doi:10.7939/r3-2qne-5s44

Hybrid Cellular Automaton for Field Cancerization

2022· dissertation· en· W6921983573 on OpenAlexaboutno aff

Bibliographic record

VenueUniversity of Alberta Library · 2022
Typedissertation
Languageen
FieldMathematics
TopicMathematical Biology Tumor Growth
Canadian institutionsnot available
Fundersnot available
KeywordsField cancerizationCarcinogenCellular automatonCancerField (mathematics)Carcinogenesis

Abstract

fetched live from OpenAlex

Field Cancerization is a hypothesis for the formation of cancer in certain types of tissues. It proposes the idea that a tumour can form in a “field” of cells that are predetermined for the development of cancer. Further, it is hypothesized that these fields are mainly caused by the onslaught of carcinogens on the tissue. Lastly, field cancerization proposes that tumour recurrence is related to the tumour being excised without fully removing the surrounding field. The model we propose is a hybrid cellular automaton (CA) used to verify the previously stated propositions and determine how long cancer development will take. The CA is considered to be hybrid due to its’ rule depending on the results of partial differential equations (PDEs) and a multi-layer perceptron (MLP). The PDEs represent the spread of carcinogens in the tissue, while the MLP computes the effects of the carcinogens on the gene expression of the genes related to cancer development in the tissue under consideration. We apply the model to field cancerization of the tongue. Most of the parameters of the model were chosen and are not based upon real data, as the necessary data was not available. This includes the choice of substituting nicotine, which is a mutagen but not currently listed as a carcinogen, to represent the carcinogen impacts of smoking tobacco. According to Health Canada tobacco contains over 4,000 chemicals, of which more than 70 are carcinogens. Many researchers are investigating how nicotine contributes to the development of cancer due to its use in non-tobacco products such as e-cigarettes and nicotine patches. One such study by Sanner & Grimsrud suggests that nicotine has several cancer-causing effects including speeding up cell growth, it is poisonous to cells, it kick-starts a process that is an important step in the path toward malignant cell growth, and it decreases the tumour suppressor CHK2. Therefore, we considered the readily available data with regards to nicotine as an appropriate choice to substitute for the over 70 carcinogens in tobacco. The other carcinogen considered in this thesis is ethanol to represent alcohol consumption. It was found that nicotine was a more potent carcinogen than ethanol. The combined impact of both ethanol and nicotine resulted in more aggressive cancer growth. It was verified that removing the field results in recurrence taking longer to occur than if the field is not removed. We also tracked cell lineages and found that as the field develops, the number of distinct cell lines decreases. Finally, we found that most tumour masses formed via polyclonal origin instead of monoclonal origin, though both occur within the simulations.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesInsufficient payload (model declined to judge)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Theoretical or conceptual · Consensus signal: Theoretical or conceptual
GenreCandidate signal: Empirical · Consensus signal: none
Teacher disagreement score0.602
Threshold uncertainty score0.992

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0090.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.010
GPT teacher head0.223
Teacher spread0.213 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designTheoretical or conceptual
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2022
Admission routes1
Has abstractyes

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