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Record W7014744781

The regulation of Pannexin1 and Pannexin2 in the skin in health and disease

2021· article· en· W7014744781 on OpenAlexfundno aff

Bibliographic record

VenueScholarship@Western (Western University) · 2021
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicConnexins and lens biology
Canadian institutionsnot available
FundersNatural Sciences and Engineering Research Council of CanadaCanadian Institutes of Health Research
KeywordsMelanomaKeratinocyteParacrine signallingIn vivoIn vitroIntracellularCellDownregulation and upregulation
DOInot available

Abstract

fetched live from OpenAlex

Pannexins (PANX1, 2, 3) are a family of channel-forming glycoproteins that mediate intracellular and paracrine signaling. In contrast to PANX2, PANX1 has been extensively investigated in the skin, modulating cell differentiation, wound healing, and melanoma development. PANX1 and PANX2 can co-exist in the same cell and form mixed channels where their glycosylation seems to regulate their intermixing. N-glycosylation and caspase cleavage have been proposed as modulators of the function of PANX1, but their effects on PANX2 are unknown. We explored the PANX2 expression in mouse skin and showed that a Panx2 splice variant (PANX2-202) is continuously expressed throughout aging skin. Furthermore, PANX2 was detected in keratinocytes and is upregulated during their in vitro differentiation. We showed that in UVB-induced apoptotic keratinocytes, caspase-3/7 cleaves the PANX2 C-terminus at residue D416. Notably, CRISPR-Cas9-mediated deletion of PANX2 delays but does not impair keratinocyte apoptosis, and its caspase-mediated cleavage is not required for this process. Thus, we propose that PANX2 promotes keratinocyte death after UVB, which may contribute to skin homeostasis. Moreover, we showed that N-glycosylation occurs at the N86 residue of PANX2, regulating folding and cell surface trafficking but not its interaction with PANX1. As PANX1 is known to modulate in vitro and ex vivo tumorigenicity of melanoma cells, we examined the effect of a germline deletion on in vivo melanoma progression by crossing Panx1 knockout (Panx1-/-) mice with the melanoma model: BrafCA, PtenloxP, Tyr::CreERT2 (BPC). We found that Panx1-deletion did not reduce melanoma formation or improve BPC-mice survival. However, tumors in BPC-Panx1-/- mice exhibited increased infiltration of CD4+, CD8+ T lymphocytes, and Granzyme B+ cells but not immunosuppressive FoxP3+ T cells. Remarkably, splenomegaly was also found in female BPC-Panx1-/- mice compared to males. Overall, this study revealed the location of two post-translational modifications in the PANX2 amino acid sequence modulating its localization and possibly its biological function. We provided further evidence that regulation of pannexins in the skin may influence cell death and the activity of the immune system during skin cancer conditions, which may have translational application in improving checkpoint inhibitor immunotherapy for melanoma.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.044
GPT teacher head0.299
Teacher spread0.255 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2021
Admission routes1
Has abstractyes

Explore more

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