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Record W7014880104

The Regulation of Beta-catenin Function in Vascular Smooth Muscle Cells

2017· other· en· W7014880104 on OpenAlexaff

Bibliographic record

VenueYork University Digital Library (York University) · 2017
Typeother
Languageen
Field
Topic
Canadian institutionsYork University
Fundersnot available
KeywordsVascular smooth muscleNeointimaMef2Wnt signaling pathwayDownregulation and upregulationTranscription factorMyocyteMEF2CEnhancerCell
DOInot available

Abstract

fetched live from OpenAlex

Vascular smooth muscle cells (VSMCs) maintain the ability to switch between a quiescent and proliferative phenotype based on external stimuli; this is an important feature for vascular repair. However, it leaves VSMCs predisposed to be affected by aberrant stimuli which results in improper cell proliferation, contributing to cardiovascular disease. Therefore, understanding the molecular mechanisms involved in activating VSMCs is of great clinical and therapeutic importance. \n \n-catenin is a multifunctional protein that plays an essential role in cell adhesion at the membrane and is required for Wnt dependent target gene expression in the nucleus. -catenin has been shown to play a significant role in VSMCs during both development and wound repair, and -catenins role is wholly dependent on specific protein-protein interactions in the cell, thus the aim of this project was to identify novel interacting partners and determine their effect on -catenin function in VSMCs. \n \nOur lab has previously identified the myocyte enhancer factor 2 (MEF2) family of transcription factors as potential -catenin interacting partners. The MEF2s are involved in vascular development during embryogenesis, but are also upregulated in the neointima following carotid artery injury in mice. In the first study, a direct interaction between MEF2 and -catenin was documented. This interaction was enhanced by p38 phosphorylation of MEF2, which resulted in increased nuclear retention of -catenin. Active p38 in primary VSMCs was able to stimulate Wnt signalling in a MEF2-dependent manner, and the MEF2--catenin interaction also synergistically increased cell proliferation. \n \nThe purpose of the second study was to identify novel -catenin interacting partners in VSMCs using mass spectrometry. The investigation revealed 131 interactions, and gene ontology enrichment analysis emphasized translation as a significant biological process. Examination of one potential interactor, the fragile X mental retardation protein (FMRP), revealed a direct interaction with -catenin. siRNA silencing of -catenin increased global protein synthesis, and biochemical analysis determined that -catenin associates at the pre-initiation complex with FMRP. Wnt-dependent nuclear shuttling of -catenin de-repressed protein translation, and we concluded that cytoplasmic -catenin can inhibit translation. Thus, we established a completely novel role for -catenin function in the regulation of translation by interaction with FMRP.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.010
GPT teacher head0.152
Teacher spread0.142 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2017
Admission routes1
Has abstractyes

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