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Record W7062024168

The role of Nix in calcium signaling, gene expression, and oxidative phenotype of skeletal muscle

2024· dissertation· en· W7062024168 on OpenAlexfundno aff

Bibliographic record

VenueMspace (University of Manitoba) · 2024
Typedissertation
Languageen
FieldEngineering
TopicThermal Analysis in Power Transmission
Canadian institutionsnot available
FundersNatural Sciences and Engineering Research Council of CanadaUniversity of Manitoba
KeywordsMitophagySkeletal muscleMediatorInsulin resistanceMitochondrionInsulin receptorRYR1MyocyteInsulin
DOInot available

Abstract

fetched live from OpenAlex

Mitochondrial quality control is an essential component of muscle biology, with multiple muscular diseases/pathologies associated with dysregulation of this process (e.g., sarcopenia, cachexia, mitochondrial myopathies, and the remodeling associated with obesity and insulin resistance). The protein Nix serves an important role as a mitophagy receptor protein that mediates removal of dysfunctional fragments of mitochondria through the autophagic process. Human genome-wide association studies have connected polymorphisms in the Nix gene with development of mitochondrially-dependent disorders such as schizophrenia and cognitive decline. In muscle, Nix expression has been connected with aging and has been implicated in starvation-induced muscle atrophy. Additionally, Nix has also been implicated as an important factor in models of lipid-induced insulin resistance of muscle, as well as a potential mediator of calcium signaling within the cell. Together this demonstrates the complex, but important, biology of Nix. However, a model to study Nix in skeletal muscle is lacking. To advance our understanding of Nix in physiologically healthy muscle, we generated and characterised a mouse line harbouring a muscle-specific deletion of Nix. I found evidence of compensated mitochondrial myopathy within muscle that manifests as reduced metabolic rate and capacity for aerobic exercise. Intriguingly, I found that Nix also regulates calcium signaling within muscle to produce a shift towards expression fast fiber types when Nix is deleted in muscle. Previously in cell culture experiments, pharmacological inhibition of Nix was found to restore insulin sensitivity following lipotoxicity-induced insulin resistance. With muscle knockout of Nix, I found heightened sensitivity to insulin that was associated with an mTOR/S6K mechanism described previously in cells. Given the variety of roles described for Nix in muscle, I took the first steps towards uncovering muscle-specific regulation of Nix expression through in vitro studies. To this end, I found that Nix is a direct target of the transcription factors MEF2D of the myogenic program. In summary, I found that deletion of Nix has multiple possible protective effects including protecting metabolism from lipid overload while also directing muscle oxidative phenotype. I have characterized a model to study the biology of Nix, which in addition to novel findings related to calcium and myostatin signaling, provides the basis to study Nix in diseases of muscle or diseases that etiologically involve muscle.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.214
Threshold uncertainty score0.998

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.006
GPT teacher head0.191
Teacher spread0.186 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2024
Admission routes1
Has abstractyes

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