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Record W7117105378 · doi:10.1002/alz70855_106266

Covid‐19 Spike protein impact on Alzheimer's (AD) and Cerebrovascular Disease

2025· article· en· W7117105378 on OpenAlexaff
Sally A. Frautschy, Kapil Manglani, Xiaohong Zuo, Cansheng Zhu, Alex Bombino, Johnathan Kwon, Mychica Jones, Douglas D. Fraser, Gregory M. Cole

Bibliographic record

VenueAlzheimer s & Dementia · 2025
Typearticle
Languageen
FieldMedicine
TopicLong-Term Effects of COVID-19
Canadian institutionsWestern University
Fundersnot available
KeywordsSpike (software development)Complement (music)Complement systemSynapseDiseaseVascular diseaseCentral nervous system

Abstract

fetched live from OpenAlex

BACKGROUND: Most COVID-19 cases fully recover, but 7% report persistent symptoms (Long Covid, LC). SARS-CoV2 uses surface S1 Spike protein to bind ACE2 receptors leading to sustained complement activation. S1 activates endothelial C3, leukocyte attachment and invasion, also observed in atherosclerosis, cerebral amyloid angiopathy (CAA), and hypertension-driven small vessel disease (CSVD). LC patients have elevated plasma AD biomarkers and postmortem evidence of microglial, complement and microvascular pathology. AD patients are at increased risk for Neuro-Covid particularly those with ApoE4 and hypertension. Here we explore modulatory roles of hypertension or ApoE4 on the response to S1 in AD rats. METHOD: We created rat models with AD or mixed AD (AD with CSVD) with or without ApoE4. Outcomes included executive function, AD pathology, white matter damage and complement activation 6 weeks after subcutaneous injection of recombinant S1. We developed high throughput plasma assays to assess vascular and CNS complement activation from rat models or human Neuro-Covid. RESULT: Experiment 1: Independent of human ApoE or hypertension, S1 increased plaque and vascular amyloid and amyloid responsive ptau217, microvascular active C3 (C3d) and neuronal and vascular complement C5b-9 terminal attack complex. E4 and hypertension synergized to increase vascular pathology induced by spike. This was accompanied by leukocyte adhesion, including neutrophils. Experiment 2: S1 spike injected 11-13 month rats showed spike-induced executive function deficits and significant increases in the hypertensive FAD rats for beta amyloidogenic APP BACE processing, ptau217 as well as loss of myelin basic protein, an index of white matter damage. We also observed increased cortical active C3, CSF C5a (ELISA), and active C3 in plasma endothelial and astrocyte-derived EV. Pre-existing hypertensive comorbidity potentiated Covid spike effects. The plasma of LC patients with Neuro-Covid showed elevated active C3 in astrocyte and endothelial EV. CONCLUSION: Spike protein caused robust increases in AD pathology as well as persistent pathogenic CNS and vascular complement activation, associated with synapse loss and BBB damage in rats with pre-existing AD and vascular pathology, a situation found in at least 20 to 30% of our aged population. Scalable assays for Neuro-Covid complement activation should facilitate treatment.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.006

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.017
GPT teacher head0.318
Teacher spread0.300 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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