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Record W7117692053 · doi:10.64898/2025.12.30.25343229

Individuals whose phenotype deviates from genetic expectation defined by common variation are enriched for rare damaging variants in genes that cause rare disease

2025· article· en· W7117692053 on OpenAlexaff
Nikolas Alessandro Baya, Frederik H. Lassen, Barney Hill, Samvida Sudheesh Venkatesh, Hannah Currant, Cecilia M. Lindgren, Duncan Stuart Palmer

Bibliographic record

VenuemedRxiv · 2025
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicGenetic Associations and Epidemiology
Canadian institutionsDiscovery Centre
Fundersnot available
KeywordsDiseaseAllelePhenotypeRare eventsRare diseaseGeneContext (archaeology)Genetic testingGenetic variationMissense mutation

Abstract

fetched live from OpenAlex

Abstract Polygenic scores (PGS) predict complex traits and stratify disease risk but often fail to fully capture individual-level variation. “Misaligned” individuals, whose observed phenotypes deviate from their genetically expected values based on polygenic scores (PGS), provide a powerful model for identifying factors beyond common-variant effects, including additional genetic factors. Here, we apply misalignment classification and enrichment testing frameworks to seven continuous and three dichotomous traits, assessing whether misaligned individuals in the UK Biobank are enriched for rare (minor allele frequency (MAF) < 0.1%) damaging genetic variation. We identify significant enrichment (false discovery rate (FDR)-adjusted P < 0.05) of predicted loss-of-function (pLoF) variants in COPB2 and GORAB among individuals misaligned for lower-than-expected bone mineral density. We refine previously observed grouped-gene enrichment in individuals with misaligned stature to the single-gene level: shorter-than-expected individuals are enriched for pLoF variants in ACAN and IGF1 , and taller-than-expected individuals are enriched for predicted damaging missense in FBN1 . Using an individual’s misalignment classification as a phenotype, we perform an exome-wide scan across seven traits, resulting in 74 FDR- significant genes. We identify KANK1 as a gene associated with later age at menopause, potentially protective against primary ovarian insufficiency. For dichotomous disease status traits, we demonstrate evidence for the liability threshold model in the context of counteracting conditionally-orthogonal common and rare variant pathogenic/protective effects. Among individuals diagnosed with type 2 diabetes, carriers of rare pathogenic pLoF variants in HNF1A and HNF4A had significantly lower polygenic risk than non- carriers (FDR-adjusted one-sided t -test P < 5 × 10 − 3 ). We also show that coronary artery disease controls carrying rare protective pLoF variants in ANGPTL3 had nominally higher polygenic risk (one-sided t -test P = 0.03) than non-carriers. This study highlights the power of misalignment-based analyses in complex continuous phenotypes and disease, with the potential to validate known genetic contributors to traits and identify novel genes. This work paves the way for better molecular diagnoses and targeted therapeutic discovery.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.004
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.010

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.004
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.001
Bibliometrics0.0010.001
Science and technology studies0.0010.001
Scholarly communication0.0010.000
Open science0.0000.001
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0030.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.017
GPT teacher head0.273
Teacher spread0.256 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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