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Record W7132998705

The role of platelet-derived growth factor-BB signaling in glycosaminoglycan formation in fetal lung cells

2002· dissertation· W7132998705 on OpenAlexfundno aff
Nicholas Julian Cartel

Bibliographic record

VenueTSpace · 2002
Typedissertation
Language
FieldMedicine
TopicNeonatal Respiratory Health Research
Canadian institutionsnot available
FundersCanadian Institutes of Health Research
KeywordsProtein kinase BPhosphorylationWortmanninPI3K/AKT/mTOR pathwaySignal transductionProteoglycanHeparan sulfateGlycosaminoglycan
DOInot available

Abstract

fetched live from OpenAlex

Elucidation and genetic determination of the signal transduction pathway(s) that results in platelet-derived growth factor (PDGF-BB)-induced glycosaminoglycan (GAG) synthesis as well as protection from apoptosis were undertaken. Over-expression of a soluble PDGF β-receptor as well as a mutated form of the β-receptor, unable to bind PI-3K, diminished GAG synthesis in fetal lung fibroblasts subsequent to PDGF-BB stimulation. Treatment with the PI-3K inhibitor, wortmannin or expression of dominant-negative PI-3K abrogated Akt activity and GAG synthesis. Furthermore, expression of dominant-negative Akt (Akt-AAA) abrogated endogenous Akt activity, Rab3D phosphorylation and GAG synthesis whereas expression of constituitively activated Akt (Akt-DD) stimulated Rab3D phosphorylation and GAG synthesis in the absence of PDGF-BB. Over-expression of wild-type PTEN inhibited Akt activity and concomitantly attenuated GAG synthesis in fibroblasts stimulated with PDGF-BB. These data suggest that Akt is an integral protein involved in PDGF-BB-mediated GAG regulation in fetal lung fibroblasts. As the Akt pathway has been implicated in cell survival mechanisms, it was investigated whether the pathway regulating GAG secretion was anti-apoptotic. Transfection of constituitively-active Akt into fetal lung fibroblasts lead to an increase in GAG secretion, without PDGF-BB stimulation, that safeguarded the cells from apoptosis. Separate cultures of lung fibroblasts were treated with xyloside, a proteoglycan substitute, or with topologically applied GAGs. Application of xyloside or the sulfated GAGs, chondroitin sulfate or heparan sulfate, resulted in an increase in GAG secretion, diminished apoptosis as well as an increase in BAD phosphorylation and diminished caspase cleavage, all of which are indicative of an anti-apoptotic response. PDGF-BB also increased p42 mitogen-activated protein kinase (MAPK), although it was determined not to be a component of the GAG pathway. However, MAPK is an enzyme that, together with Akt, phosphorylates and sequesters BAD. Overexpression of a dominant-negative PDGFβ-receptor, lacking the cytoplasmic signaling domain, or a dominant-negative PDGFβ-receptor, with cytoplasmic tyrosines mutated to phenylalanine, abrogated p42MAPK activity suggestive that PDGF-BB mediated activation of p42MAPK requires the PDGFβ-receptor but is independent of its tyrosine phosphorylation. The data contained herein are demonstrative of a concerted and pleiotropic response initiated by the GAG signal transduction pathway as a means to ensure appropriate fetal lung development.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.030
GPT teacher head0.357
Teacher spread0.327 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2002
Admission routes1
Has abstractyes

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