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Record W7161780451 · doi:10.82308/27347

Role of the obese adipose tissue microenvironment in cancer progression

2024· dissertation· en· W7161780451 on OpenAlexaboutno aff
Laura Wu

Bibliographic record

Venuenot available
Typedissertation
Languageen
FieldMedicine
TopicAdipose Tissue and Metabolism
Canadian institutionsnot available
Fundersnot available
KeywordsThermogenesisAdipose tissueWhite adipose tissueTumor microenvironmentMetaboliteBrown adipose tissueObesityCancer

Abstract

fetched live from OpenAlex

Background: Obesity is associated with a greater risk of developing cancer and a higher mortality rate, which can in part be attributed to the influence of the adipose tissue microenvironment (ATME). White adipose tissue (WAT) stores energy but accumulates in obesity to cause chronic inflammation. On the other hand, brown adipose tissue (BAT) produces heat from chemical energy through thermogenesis. Although BAT is scarce in adults, WAT can develop BAT-like characteristics through a process called “browning” upon exposure to thermogenic stimuli. Although BAT and WAT are known to influence metabolite availability and nutrient composition of tissues, little is known about how the obese ATME can influence metabolite availability within the tumor microenvironment. Our preliminary data showed that obesity (i.e. elevated WAT) promotes tumor growth, while thermogenesis (i.e. WAT browning) blunts tumor progression in lean and obese mice using a melanoma model. Therefore, the objective of this project was to identify how the obese ATME promotes cancer progression in additional cancer models, and whether thermogenesis can be used to offset obesity-driven cancer. Methods: Female C57BL6 mice were placed on a high-fat diet or low-fat diet for 15 weeks and injected with tumor cells in the flank or mammary fat pads. Thermogenesis was induced with a β3-adrenoceptor agonist (CL-316,243) or cold exposure. Transgenic female MMTV-PyMT mice were housed at three temperatures (6.5°C, 22°C, 26.5°C; collaboration with the Kazak lab) to simulate cold, room temperature, and thermoneutral conditions, respectively. Tumor interstitial fluid (TIF) from lean and obese mice was analyzed by liquid chromatography–mass spectrometry to study changes in metabolite availability within the tumor microenvironment. Results: Consistent with previous data in breast cancer (E0771, PyMT) and melanoma (B16F10) models, obesity was sufficient to accelerate tumor growth in the MC-38 (colorectal cancer) and Pan02 (pancreatic ductal adenocarcinoma) models. However, unlike the melanoma model, treatment with CL-316,243 had no effect on tumor growth. Cold exposure also had no effect on tumor growth in the MMTV-PyMT (breast cancer) model. To further explore how obesity contributes to accelerated tumor growth, a protocol for isolating and extracting TIF was optimized (collaboration with the McGill Metabolomics Core). Trends in TIF metabolite availability were found and differed from the content of blood plasma between lean and obese mice. Conclusions: This research may provide a better understanding of the underlying mechanisms that contribute to obesity-driven cancer, and suggests that changes in metabolite availability may in part underlie differences in tumor progression between lean and obese preclinical models

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.004

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.009
GPT teacher head0.314
Teacher spread0.306 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2024
Admission routes1
Has abstractyes

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