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Record W7162037200 · doi:10.82308/274

Understanding the TGF-beta language or getting lost n translation: The TGFbeta microRNAome

2019· dissertation· en· W7162037200 on OpenAlexaboutno aff
Nadège Fils-Aimé

Bibliographic record

Venuenot available
Typedissertation
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicMicroRNA in disease regulation
Canadian institutionsnot available
Fundersnot available
KeywordsGene silencingmicroRNAContext (archaeology)Breast cancerCancerRegulation of gene expressionFunction (biology)Gene expressionCell growth

Abstract

fetched live from OpenAlex

Breast cancer is the most commonly diagnosed female cancer in the world. Consequently, researchers deployed tremendous efforts that led to a significant increase in life expectancy and quality of life. Despite those advances, breast cancer remains the second leading cause of female cancer-related deaths after lung cancer in Canada. Considering their cost-effective nature, microRNAs (miRNAs) have become an interesting therapeutic avenue in the treatment of diseases, including cancer. Micro-RNAs are small, non-coding RNAs whose main function is in regulating the output of RNA translation. By binding the 3’UTR of the messages they target, miRNAs add a level of regulation that ensures homeostasis is maintained. In the advent of cancer, the miRNA expression profile is dysregulated, this being the cause for, or the consequence of, expression of genes that favour tumour progression and the silencing of those that protect the cell from tumorigenesis.In this thesis, we examined the regulation of miRNAs in the context of TGFbeta-mediated mammary tumour suppression and progression. This cytokine behaves as a tumour suppressor, a role commonly observed in healthy epithelial cells and early carcinoma but lost to benefit a pathway that signals for tumour progression in more advanced cancers. Interestingly, TGF also regulates the expression of a number of miRNAs. Here, we show that TGFbeta negatively regulates the expression of the miR-30 family of miRNAs. We found them to antagonize TGFbeta-mediated tumour suppression by preventing the modulation of the expression of genes important for cell apoptosis, cytostasis, and inhibition of immortalization. We found the miR-30s are more highly expressed in basal breast cancer, where TGFbeta lost its tumour suppressive function, and that their inhibition impedes tumour development in vivo. Moreover, we found another down-regulated miRNA, miR-584, to prevent TGFbeta-mediated cell migration. This miRNA functions by preventing the expression of the actin-binding protein PHACTR1, leading to a reorganization of the cytoskeleton that prevents cell motility. Thus, we found a way to control both the tumour-suppressor and pro-metastatic roles of TGFbeta.With this research, we have elucidated the involvement of miRNAs downstream of TGFbeta in the development of breast cancer, and potentially opened the door for the development of a TGFbeta-targeting therapy

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: none
Teacher disagreement score0.002
Threshold uncertainty score0.005

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.002
Scholarly communication0.0020.004
Open science0.0000.001
Research integrity0.0010.003
Insufficient payload (model declined to judge)0.0010.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.040
GPT teacher head0.287
Teacher spread0.247 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2019
Admission routes1
Has abstractyes

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