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Enregistrement W2052999954 · doi:10.1158/1538-7445.panca2012-b13

Abstract B13: Whole-genome mutation landscape in pancreatic ductal adenocarcinoma.

2012· article· en· W2052999954 sur OpenAlexaff
Carson Holt, Fouad Yousif, Lee E. Timms, Michelle Sam, Kimberly Begley, Thomas J. Hudson, John D. McPherson, Lincoln Stein, Lakshmi Muthuswamy, Christina K. Yung, Tim N. Beck, Bojan Losic, Niloofar Arshadi, Christine Ouelltt, Irinia Kalatskaya, Richard de Borja, Robert E. Denroche

Notice bibliographique

RevueGenetics · 2012
Typearticle
Langueen
DomaineBiochemistry, Genetics and Molecular Biology
ThématiqueCancer Genomics and Diagnostics
Établissements canadiensOntario Institute for Cancer Research
Organismes subventionnairesnon disponible
Mots-clésIndelGenomeSomatic cellBiologyCopy-number variationAdenocarcinomaCopy number analysisCancerPancreatic cancerPrimary tumorStructural variationComputational biologyCancer researchGeneticsGeneSingle-nucleotide polymorphismMetastasisGenotype

Résumé

récupéré en direct d'OpenAlex

Pancreatic ductal adenocarcinoma (PDAC) is a rare cancer with a very high mortality rate. Because it is extremely difficult to detect at an early stage; PDAC tumors often spread to regional lymph nodes or distant metastases by the time they are diagnosed. Published reports have already identified a number of chromosomal alterations at many genomic levels; however PDAC still lacks a comprehensive catalogue for the whole genome mutation spectrum. The goal of our study is to annotate all types of identifiable genomic aberrations based on whole genome sequencing of 5 PDAC tumors. It is a common knowledge that surgical primary tissues of PDAC have very low tumor content. Hence, for our study all five primary tumors have been modeled as xenografts using NOD-SCID mice to enrich for tumor cells. Here, we report on the cancer-specific genome alterations in 5 PDAC tumors and show that xenograft models do represent genomic landscape of primary tumors. All samples were whole-genome sequenced using Illumina HiSeq to give a minimum coverage of 30X. We have developed an analysis pipeline to identify somatic single nucleotide variations (SNVs) using The Genome Analysis Tool Kit (GATK), copy number alterations (CNAs) using KSseg (in-house CNV algorithm) and structural variations using Geometric Analysis of Structural Variants (GASV). A number of filters have been implemented to separate germline variants and mouse derived contamination from the cancer specific somatic variation. Our analysis has identified an average of 1527 SNVs, 1555 INDELs, and 53 CNAs per PDAC genome (combined for primary and xenograft). All somatic SNVs were verified using Ion Torrent based sequencing technology with a verification rate of 93%. CNAs were verified using Nimblegen 2.1M Array-based Comparative Genome Hybridization technology, and produced a verification rate of greater than 98% for losses and 60-97% for gains. We also observed a high level of overlap between primary tumor and xenograft samples, with 84% of total primary tumor SNVs and 61% of INDELS (called across all samples) being found in the correlating xenograft genome. After verification of SNVs by deep sequencing, we observe an additional 50% of SNVs that were called only in the xenograft samples validate in the primary sample. Our results show that the somatic single nucleotide mutation rate is in the range of 1 - 4 SNVs/Mb and there is a statistically significant increase in the G>T transversions. It is well known that methylated CpG dinucleotides are the preferred sites for G > T transversions and we are investigating the role played by DNA methylation alterations. All somatic variants were annotated using an in-house software package based on Sequence Ontology classification of variant effects to integrate different types of variations and provide a functional interpretation. Our analysis has identified 290 genes that are functionally impacted in 4 or more genomes by any type of mutation. They include 6 known oncogenes, 10 protein kinases, 9 cell differentiation markers, 17 transcription factors and 6 cytokines and growth factors. Functional enrichment analysis on this gene set using MSigDB v3.0 database shows important cancer-related pathways including the NK cells pathway, the Adherens junctions interactions pathway, and the axon guidance signaling pathway. Citation Format: Carson Holt, Fouad Yousif, Lee Timms, Michelle Sam, Kimberly Begley, Thomas Hudson, John D. McPherson, Lincoln D. Stein, Lakshmi B. Muthuswamy, Christina Yung, Tim Beck, Bojan Losic, Niloofar Arshadi, Christine Ouelltt, Irinia Kalatskaya, Richard de Borja, Robert Denroche. Whole-genome mutation landscape in pancreatic ductal adenocarcinoma. [abstract]. In: Proceedings of the AACR Special Conference on Pancreatic Cancer: Progress and Challenges; Jun 18-21, 2012; Lake Tahoe, NV. Philadelphia (PA): AACR; Cancer Res 2012;72(12 Suppl):Abstract nr B13.

Récupéré en direct depuis OpenAlex et désinversé. Les résumés ne sont pas conservés dans cette base de données : les index inversés représentent 8,6 Go des 9,3 Go de texte de la base, et le serveur dispose de 13 Go libres.

Comment cette classification a été obtenuedéplier

Prédiction distillée sur la base complète

Imitation des enseignants

Ni prévalence calibrée, ni vérité terrain. Validation humaine à venir. Apprise à partir de 10 348 étiquettes directes de Codex et de 10 348 étiquettes directes de Gemma. Le mode candidate est l'union des têtes enseignantes seuillées; le consensus est leur intersection. Ces sorties portent le statut machine_predicted_unvalidated et ne sont ni des étiquettes humaines ni des étiquettes directes de modèles de pointe.

score de la tête « metaresearch » (Codex)0,000
score de la tête « metaresearch » (Gemma)0,000
Version: codex-gemma-dda1882f352aStatut de validation: machine_predicted_unvalidated
Catégories candidatesaucune
Catégories consensuellesaucune
DomaineSignal candidat: aucune · Signal consensuel: aucune
Devis d'étudeSignal candidat: Observationnel · Signal consensuel: Observationnel
GenreSignal candidat: Empirique · Signal consensuel: Empirique
Score de désaccord entre enseignants0,403
Score d'incertitude au seuil0,694

Scores Codex et Gemma par catégorie

CatégorieCodexGemma
Métarecherche0,0000,000
Méta-épidémiologie (sens strict)0,0000,000
Méta-épidémiologie (sens large)0,0000,000
Bibliométrie0,0000,000
Études des sciences et des technologies0,0000,000
Communication savante0,0000,000
Science ouverte0,0000,000
Intégrité de la recherche0,0000,000
Charge utile insuffisante (le modèle a refusé de juger)0,0000,000

Scores machine (provisoires)

Les deux têtes enseignantes du modèle étudiant, lues sur ce travail. Un score ordonne la base pour la relecture; il n'affirme jamais une catégorie, et le statut de validation accompagne chaque rangée tel quel.

Scores de référence d'un modèle non mature (critères de maturité non atteints, 7 itérations). Un score ordonne; il n'affirme jamais une catégorie.

Tête enseignante Opus0,010
Tête enseignante GPT0,235
Écart entre enseignants0,225 · la distance entre les deux têtes enseignantes sur ce seul travail
Statut de validationscore_only:v0-immature-baseline · tel quel depuis la passe de notation : score_only signifie que le nombre peut ordonner les travaux, et qu'aucune étiquette de catégorie n'en découle

Classification

machine, non validée

Prédiction automatique; un appel candidat d’une seule tête enseignante, pas un consensus.

Les modèles n’ont appliqué aucune catégorie : rien dans la taxonomie ne correspondait à ce travail.
Devis d'étudeObservationnel
Domainenon disponible
GenreEmpirique

Le détail, modèle par modèle et score par score, se trouve en fin de page sous « Comment cette classification a été obtenue ».

En bref

Citations0
Publié2012
Routes d'admission1
Résumé présentoui

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