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Record W1539559867 · doi:10.1186/ar1395

Protective role of interferon gamma in murine antigen-induced arthritis

2004· article· en· W1539559867 on OpenAlexfundno aff
Marion Hückel, Ingo M. Irmler, Jutta Simon, Andreas Radbruch, Annika Scheffold, Rolf Bräuer

Bibliographic record

VenueArthritis Research · 2004
Typearticle
Languageen
FieldImmunology and Microbiology
Topicinterferon and immune responses
Canadian institutionsnot available
FundersCanadian Arthritis NetworkSchool of Medicine, University of California, San DiegoMenzies Institute for Medical ResearchNational Cancer InstituteArthritis SocietyUniversity of North Carolina at Chapel HillGenentechNational Institutes of HealthBiogenLupus Research InstitutePfizerNatural Sciences and Engineering Research Council of CanadaDutch Arthritis AssociationOesterreichische NationalbankDeutsche ForschungsgemeinschaftNuffield FoundationPhysiotherapy Foundation of CanadaCanadian Institutes of Health ResearchNational Institute of Arthritis and Musculoskeletal and Skin DiseasesLupus Research AllianceWellcome TrustNewcastle UniversityNational Institute of Allergy and Infectious DiseasesHoward Hughes Medical InstituteAustrian Science FundArthritis Foundation
KeywordsBusiness

Abstract

fetched live from OpenAlex

In human rheumatoid arthritis interactions of immune cells with resident cells, especially synovial fibroblasts, play a crucial role in the perpetuation of inflammatory and destructive processes in affected joints. Apart from macrophages, Th cells are predominant in cell infiltrates, and Th cell responses are specified as Th1 type. As interferon gamma (IFN-γ) secreting Th1 cells are suspected to promote autoimmune diseases, there is the notion that a shift of a Th1 response towards a Th2 response might ameliorate arthritis. However, in different models and therapy studies conflicting results are described. Thus, we investigated the role of IFN-γ in murine antigen-induced arthritis (AIA), a model with homologies to human rheumatoid arthritis in terms of histopathology, chronicity and responses to several immunomodulatory drugs. AIA was evaluated in IFN-γ mice in comparison with C57BL/6 wild-type mice by clinical, histological and immunological parameters. Furthermore, Th cells isolated from immunised mice were separated according to their IFN-γ on the cell surface into IFN-γ and IFN-γ Th cells and than transferred into naive mice, which received at the same time a single injection of antigen (methylated bovine serum albumin) into the knee joint for arthritis induction. In cocultures with synovial fibroblasts, the stimulatory capacity of IFN-γ and IFN-γ Th cells was tested in vitro , analysing the levels of matrix metalloproteases, IL-6, and nitric oxide in supernatants. IFN-γ mice developed a more severe arthritis than wild-type mice with higher knee joint swelling, stronger acute inflammation and increased chronic joint destruction, even though IgG levels (total and methylated bovine serum albumin-specific) were reduced. Application of rIFN-γ ameliorates the severity of AIA in IFN-γ mice. Congruently, the transfer of IFN-γ Th cells from immunised wild-type mice into naive mice led to a more severe arthritis than the transfer of IFN-γ Th cells. Furthermore, IFN-γ Th cells had a stronger stimulatory capacity for matrix metalloproteases and IL-6 in synovial fibroblasts in vitro , whereas IFN-γ Th cells were more potent nitric oxide inducers. Inducing processes are mainly driven by direct cell contact. These experiments suggest a protective role of IFN-γ in the experimental arthritis model. The lack of IFN-γ in Th cells exerted a higher disease-promoting effect in vivo as well as a higher stimulatory capacitiy to synovial fibroblasts in vitro . The recent data generated in experimental models indicate a more complex role of IFN-γ in the pathogenesis of Th1-mediated autoimmune diseases than originally proposed.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesInsufficient payload (model declined to judge)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.101
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0010.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.032
GPT teacher head0.322
Teacher spread0.290 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2004
Admission routes1
Has abstractyes

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