MétaCan
Menu
Back to cohort
Record W1975596068 · doi:10.1371/journal.pone.0108087

Genome-Wide Investigation of DNA Methylation Marks Associated with FV Leiden Mutation

2014· article· en· W1975596068 on OpenAlexafffund
Dylan Aïssi, Jessica Dennis, Vinh Trương, Nora Zwingerman, Ares Rocañín-Arjó, Marine Germain, Tara Paton, Pierre‐Emmanuel Morange, France Gagnon, David‐Alexandre Trégouët

Bibliographic record

VenuePLoS ONE · 2014
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicCancer-related molecular mechanisms research
Canadian institutionsHospital for Sick ChildrenCentre Hospitalier de l’Université de MontréalPublic Health OntarioUniversity of Toronto
FundersCanadian Institutes of Health ResearchUniversité Pierre et Marie CurieAgence Nationale de la RechercheU.S. Public Health ServiceHeart and Stroke Foundation of Canada
KeywordsDNA methylationCpG siteGeneticsSingle-nucleotide polymorphismPenetranceMethylationLocus (genetics)BiologyMutationMolecular biologyDNAGenotypeGenePhenotype

Abstract

fetched live from OpenAlex

In order to investigate whether DNA methylation marks could contribute to the incomplete penetrance of the FV Leiden mutation, a major genetic risk factor for venous thrombosis (VT), we measured genome-wide DNA methylation levels in peripheral blood samples of 98 VT patients carrying the mutation and 251 VT patients without the mutation using the dedicated Illumina HumanMethylation450 array.The genome-wide analysis of 388,120 CpG probes identified three sites mapping to the SLC19A2 locus whose DNA methylation levels differed significantly (p,3 10 28 ) between carriers and noncarriers.The three sites replicated (p,2 10 27 ) in an independent sample of 214 individuals from five large families ascertained on VT and FV Leiden mutation among which 53 were carriers and 161 were non-carriers of the mutation.In both studies, these three CpG sites were also associated (2.33 10 211 ,p,3.02 10 24 ) with biomarkers of the Protein C pathway known to be influenced by the FV Leiden mutation.A comprehensive linkage disequilibrium (LD) analysis of the whole locus revealed that the original associations were due to LD between the FV Leiden mutation and a block of single nucleotide polymorphisms (SNP) located in SLC19A2.After adjusting for this block of SNPs, the FV Leiden mutation was no longer associated with any CpG site (p.0.05).In conclusion, our work clearly illustrates some promises and pitfalls of DNA methylation investigations on peripheral blood DNA in large epidemiological cohorts.DNA methylation levels at SLC19A2 are influenced by SNPs in LD with FV Leiden, but these DNA methylation marks do not explain the incomplete penetrance of the FV Leiden mutation.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.006

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.001
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.020
GPT teacher head0.227
Teacher spread0.207 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations11
Published2014
Admission routes2
Has abstractyes

Explore more

Same venuePLoS ONESame topicCancer-related molecular mechanisms researchFrench-language works237,207