MétaCan
Menu
Back to cohort
Record W1977313215 · doi:10.1016/j.jalz.2013.05.851

P2–205: Butyrylcholinesterase‐associated neuropathology in early‐onset familial Alzheimer's disease

2013· article· en· W1977313215 on OpenAlexaff
Sultan Darvesh, Meghan K. Cash, Kenneth Rockwood

Bibliographic record

VenueAlzheimer s & Dementia · 2013
Typearticle
Languageen
FieldMedicine
TopicCholinesterase and Neurodegenerative Diseases
Canadian institutionsDalhousie University
Fundersnot available
KeywordsNeuropathologyButyrylcholinesterasePresenilinPathologyAlzheimer's diseaseThioflavinAmyloid (mycology)DementiaNeurofibrillary tangleImmunohistochemistrySenile plaquesBiologyMedicineDiseaseAchéAcetylcholinesteraseEnzyme

Abstract

fetched live from OpenAlex

Early-onset familial Alzheimer's disease (FAD) is characterized as an uncommon form of AD affecting genetically predisposed individuals before age 65. Details of neuropathological data concerning FAD mutations of genes encoding for presenilin-1 (PS1) are limited, particularly for the mutation Leu235Pro occurring in the TM5 domain of PS1. Butyrylcholinesterase (BuChE) is an enzyme involved in the regulation of cholinergic neurotransmission and is found in association with β-amyloid (Aβ) plaques and tau neurofibrillary tangles (NFTs) in AD. We hypothesized, as with sporadic AD, BuChE is associated with these AD neuropathological hallmarks in FAD. Post-mortem brain tissues from three FAD cases with the PS1 mutation Leu235Pro and one case with confirmed sporadic AD were examined. Adjacent sections of brain tissues were stained with Aβ and tau immunohistochemical methods and thioflavin-S and BuChE histochemical methods. All brains were given an “ABC” score using the National Institute on Aging-Alzheimer's Association neuropathology guidelines for AD. Plaque loads in select regions were quantified using NIH ImageJ software and recorded as a percentage of the total area. All cases demonstrated severe dementia (Global Deterioration Scale scores = 6) prior to death. AD pathology was prominent throughout the rostrocaudal extent of all four brains; ABC scores revealed high neuropathological change. In FAD cases, the distribution of neuronal BuChE paralleled that in normal human tissue and was associated with Aβ plaques and NFTs, as previously described in sporadic AD. Of note was the abundance of Aβ-, BuChE- and thioflavin-S-positive plaques. Plaque density was most notable with Aβ staining, followed by subsequent decreases of BuChE and thioflavin-S plaque staining. The Leu235Pro substitution of the PSEN1 gene produces significant behavioral and pathological impairment, with onset typically before 40 years of age. As reported in cases of sporadic AD, these findings confirm that BuChE is highly associated with AD pathology in this mutation and may be involved in the maturation of toxic Aβ protofibrils. This enzyme may hold promise as a therapeutic and neuroimaging target in AD, particularly in cases of aggressive FAD forms.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.004
Threshold uncertainty score0.014

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.001
Science and technology studies0.0010.000
Scholarly communication0.0010.000
Open science0.0000.000
Research integrity0.0010.000
Insufficient payload (model declined to judge)0.0040.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.031
GPT teacher head0.285
Teacher spread0.254 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2013
Admission routes1
Has abstractyes

Explore more

Same venueAlzheimer s & DementiaSame topicCholinesterase and Neurodegenerative DiseasesFrench-language works237,207