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Record W1980420968 · doi:10.1158/1538-7445.am2011-4703

Abstract 4703: Mitochondrial DNA mutations affect respiratory complex-I in never-smoker lung cancer patients and contribute to lung cancer cell growth

2011· article· en· W1980420968 on OpenAlexaff
Santanu Dasgupta, Ethan Soudry, Chunbo Shao, Nitai D. Mukhopadhyay, John Yee, Stephen Lam, Wan L. Lam, Paul B. Fisher, David Sidransky

Bibliographic record

VenueCancer Research · 2011
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicRNA modifications and cancer
Canadian institutionsUniversity of British Columbia
Fundersnot available
KeywordsMitochondrial DNALung cancerCancerMedicineLungInternal medicineOncologySomatic cellGenotypeMitochondrionPathologyGastroenterologyBiologyGeneticsGene

Abstract

fetched live from OpenAlex

Abstract Lung cancer is a deadly disease worldwide with smoking being the most important risk factor. In the United States, it is estimated that there will be over 222,550 cases of lung cancer resulting in 157,300 deaths in 2010. Despite significant improvement in therapeutic modalities, the overall 5-year survival rate is still only 15%. The high morbidity is due to the lack of early detection and therapeutic intervention. Other than nuclear genetic alterations, mitochondrial DNA mutations were reported in different human cancers and implicated to have a potential role in biomarker development. However, mtDNA alteration patterns and their potential involvement are unknown in lung cancer patients that have never smoked. In the present study, we sequenced the entire mitochondrial genome and measured the mtDNA content index by quantitative PCR in 60 lung cancer patients. Of the 60 patients, 30 never smoked and the remaining 30 patients were current smokers. Forty five percent of patients (27/60) displayed at least one somatic mtDNA mutation and a total of 41 mutations were detected which were mostly homoplasmic in nature. Strikingly, mtDNA mutations were significantly higher (P=0.0025) among the non-smokers as compared to the current smoking lung cancer patients and these mutations significantly affected (P=0.025) mitochondria encoded respiratory complex-I. The patients of Asian origin also had significantly higher (P=0.015) mtDNA mutations compared to the Caucasian patient populations. The mtDNA content index also significantly increased (P=0.037) in the non-smokers compared to the smoker lung cancer patients. Forced overexpression of a frequently observed mtDNA mutation from the ND5 gene of respiratory complex-I resulted in increased superoxide production, in vitro proliferation and invasion of lung cancer cells. Frequent mtDNA alterations are detectable in non-smoker lung cancer patients and may contribute to lung cancer progression. Citation Format: {Authors}. {Abstract title} [abstract]. In: Proceedings of the 102nd Annual Meeting of the American Association for Cancer Research; 2011 Apr 2-6; Orlando, FL. Philadelphia (PA): AACR; Cancer Res 2011;71(8 Suppl):Abstract nr 4703. doi:10.1158/1538-7445.AM2011-4703

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.009

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0030.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.058
GPT teacher head0.378
Teacher spread0.320 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations2
Published2011
Admission routes1
Has abstractyes

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