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Record W1999182967 · doi:10.1172/jci63606

Toll-like receptor 7 regulates pancreatic carcinogenesis in mice and humans

2012· article· en· W1999182967 on OpenAlexfundno aff
Atsuo Ochi, Christopher S. Graffeo, Constantinos P. Zambirinis, Adeel ur Rehman, Michael Hackman, Nina Fallon, Rocky Barilla, Justin R. Henning, Mohsin Jamal, Raghavendra Rao, Stephanie H. Greco, Michael Deutsch, Marco V. Medina-Zea, Usama Saeed, Melvin Ego-Osuala, Cristina Hajdu, George Miller

Bibliographic record

VenueJournal of Clinical Investigation · 2012
Typearticle
Languageen
FieldMedicine
TopicPancreatic and Hepatic Oncology Research
Canadian institutionsnot available
FundersNational Institute of Diabetes and Digestive and Kidney DiseasesNational Institutes of HealthNational Cancer InstituteSchool of Medicine, New York UniversityYork UniversityNational Pancreas Foundation
KeywordsPancreatic cancerCancer researchStromal cellCarcinogenesisBiologyTLR7Tumor microenvironmentDownregulation and upregulationPancreatic Intraepithelial NeoplasiaPancreatic tumorCancerToll-like receptorImmunologyInnate immune systemImmune system

Abstract

fetched live from OpenAlex

Pancreatic ductal adenocarcinoma is an aggressive cancer that interacts with stromal cells to produce a highly inflammatory tumor microenvironment that promotes tumor growth and invasiveness.The precise interplay between tumor and stroma remains poorly understood.TLRs mediate interactions between environmental stimuli and innate immunity and trigger proinflammatory signaling cascades.Our finding that TLR7 expression is upregulated in both epithelial and stromal compartments in human and murine pancreatic cancer led us to postulate that carcinogenesis is dependent on TLR7 signaling.In a mouse model of pancreatic cancer, TLR7 ligation vigorously accelerated tumor progression and induced loss of expression of PTEN, p16, and cyclin D1 and upregulation of p21, p27, p53, c-Myc, SHPTP1, TGF-β, PPARγ, and cyclin B1.Furthermore, TLR7 ligation induced STAT3 activation and interfaced with Notch as well as canonical NF-κB and MAP kinase pathways, but downregulated expression of Notch target genes.Moreover, blockade of TLR7 protected against carcinogenesis.Since pancreatic tumorigenesis requires stromal expansion, we proposed that TLR7 ligation modulates pancreatic cancer by driving stromal inflammation.Accordingly, we found that mice lacking TLR7 exclusively within their inflammatory cells were protected from neoplasia.These data suggest that targeting TLR7 holds promise for treatment of human pancreatic cancer. Results Elevated expression of TLR7 in pancreatic cancer.To determine the relevance of TLR7 to pancreatic cancer, we examined expression of TLR7 in the normal murine and human pancreas and in murine and human pancreatic adenocarcinoma.TLR7 expression was rare in the normal mouse pancreas.Conversely, in pancreata from 6-month-old p48Cre;Kras G12D mice, both inflammatory cells within the tumor microenvironment and ductal epithelial cells robustly expressed TLR7 (Figure 1A).We also found high expression of TLR7 in transgenic mouse models of pancreatic cancer harboring

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.005
Threshold uncertainty score0.016

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0050.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.128
GPT teacher head0.425
Teacher spread0.296 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations208
Published2012
Admission routes1
Has abstractyes

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