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Record W2007264239 · doi:10.1371/journal.pgen.1004123

Identification of Novel Genetic Loci Associated with Thyroid Peroxidase Antibodies and Clinical Thyroid Disease

2014· review· en· W2007264239 on OpenAlexaff
Marco Medici, Eleonora Porcu, Giorgio Pistis, Alexander Teumer, Suzanne J. Brown, Richard A. Jensen, Rajesh Rawal, Greet Roef, Theo S. Plantinga, Sita H. Vermeulen, Jari Lahti, Matthew J. Simmonds, Lise Lotte N. Husemoen, Rachel M. Freathy, Beverley M. Shields, Diana Pietzner, Rebecca Nagy, Linda Broer, Layal Chaker, Tim I.M. Korevaar, Maria Grazia Plia, Cinzia Sala, Uwe Völker, J. Brent Richards, Fred C.G.J. Sweep, Christian Gieger, Tanguy Corre, Eero Kajantie, Betina H. Thuesen, Youri Taes, W. Edward Visser, Andrew T. Hattersley, Jürgen Kratzsch, Alexander Hamilton, Wěi Li, Georg Homuth, Monia Lobina, Stefano Mariotti, Nicole Soranzo, Massimiliano Cocca, Matthias Nauck, Christin Spielhagen, Alec Ross, Alice M. Arnold, Martijn van de Bunt, Sandya Liyanarachchi, Margit Heier, Hans J. Grabe, Corrado Masciullo, Tessel E. Galesloot, Ee Mun Lim, Eva Reischl, Peter J. Leedman, Sandra Lai, Alessandro Delitala, Alexandra Bremner, David I. W. Philips, John Beilby, Antonella Mulas, Matteo Vocale, Gonçalo R. Abecasis, Tom Forsén, Elisabeth Widén, Jennie Hui, Holger Prokisch, Ernst E. Rietzschel, Aarno Palotie, Peter Feddema, Stephen J. Fletcher, Katharina Schramm, Jerome I. Rotter, Alexander Kluttig, Dörte Radke, Michela Traglia, Gabriela Surdulescu, Huiling He, Jayne A. Franklyn, Daniel Tiller, Bijay Vaidya, Tim De Meyer, Torben Jørgensen, Johan G. Eriksson, Peter O’Leary, Eric Wichmann, Ad R. Hermus, Bruce M. Psaty, Till Ittermann, Albert Hofman, Emanuele Bosi, David Schlessinger, Henri Wallaschofski, Nicola Pirastu, Yurii S. Aulchenko, Albert de la Chapelle, Romana T. Netea‐Maier, Stephen Gough, Henriette E. Meyer zu Schwabedissen, Timothy M. Frayling, Jean‐Marc Kaufman, Allan Linneberg, Katri Räikkönen, Johannes W. A. Smit, Lambertus A. Kiemeney, Fernando Rivadeneira, André G. Uitterlinden, John P. Walsh, Christa Meisinger, Martin den Heijer, Theo J. Visser, Timothy D. Spector, Scott G. Wilson, Henry Völzke, Anne Rentoumis Cappola, Daniela Toniolo, Serena Sanna, Silvia Naitza, Robin P. Peeters

Bibliographic record

VenuePLoS Genetics · 2014
Typereview
Languageen
FieldMedicine
TopicThyroid Disorders and Treatments
Canadian institutionsMcGill University
FundersNational Institute of Diabetes and Digestive and Kidney DiseasesNational Heart, Lung, and Blood InstituteNational Institute on AgingNational Health and Medical Research CouncilCedars-Sinai Medical CenterDeutsche ForschungsgemeinschaftNational Cancer InstituteHealthwayNational Center for Research ResourcesNewcastle UniversityFonds Wetenschappelijk OnderzoekMedical Research CouncilCardiff UniversityNational Institute of Neurological Disorders and StrokeNational Institute for Health and Care ResearchVlaamse regeringNational Center for Advancing Translational SciencesNational Human Genome Research InstituteNederlandse Organisatie voor Wetenschappelijk OnderzoekWellcome TrustHelsingin Yliopisto
KeywordsBiologyThyroidIdentification (biology)DiseaseGeneticsThyroid peroxidaseThyroid diseaseGraves' diseaseComputational biologyInternal medicineMedicine

Abstract

fetched live from OpenAlex

Autoimmune thyroid diseases (AITD) are common, affecting 2-5% of the general population. Individuals with positive thyroid peroxidase antibodies (TPOAbs) have an increased risk of autoimmune hypothyroidism (Hashimoto's thyroiditis), as well as autoimmune hyperthyroidism (Graves' disease). As the possible causative genes of TPOAbs and AITD remain largely unknown, we performed GWAS meta-analyses in 18,297 individuals for TPOAb-positivity (1769 TPOAb-positives and 16,528 TPOAb-negatives) and in 12,353 individuals for TPOAb serum levels, with replication in 8,990 individuals. Significant associations (P<5×10(-8)) were detected at TPO-rs11675434, ATXN2-rs653178, and BACH2-rs10944479 for TPOAb-positivity, and at TPO-rs11675434, MAGI3-rs1230666, and KALRN-rs2010099 for TPOAb levels. Individual and combined effects (genetic risk scores) of these variants on (subclinical) hypo- and hyperthyroidism, goiter and thyroid cancer were studied. Individuals with a high genetic risk score had, besides an increased risk of TPOAb-positivity (OR: 2.18, 95% CI 1.68-2.81, P = 8.1×10(-8)), a higher risk of increased thyroid-stimulating hormone levels (OR: 1.51, 95% CI 1.26-1.82, P = 2.9×10(-6)), as well as a decreased risk of goiter (OR: 0.77, 95% CI 0.66-0.89, P = 6.5×10(-4)). The MAGI3 and BACH2 variants were associated with an increased risk of hyperthyroidism, which was replicated in an independent cohort of patients with Graves' disease (OR: 1.37, 95% CI 1.22-1.54, P = 1.2×10(-7) and OR: 1.25, 95% CI 1.12-1.39, P = 6.2×10(-5)). The MAGI3 variant was also associated with an increased risk of hypothyroidism (OR: 1.57, 95% CI 1.18-2.10, P = 1.9×10(-3)). This first GWAS meta-analysis for TPOAbs identified five newly associated loci, three of which were also associated with clinical thyroid disease. With these markers we identified a large subgroup in the general population with a substantially increased risk of TPOAbs. The results provide insight into why individuals with thyroid autoimmunity do or do not eventually develop thyroid disease, and these markers may therefore predict which TPOAb-positives are particularly at risk of developing clinical thyroid dysfunction.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.002
metaresearch head score (Gemma)0.004
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Systematic review · Consensus signal: none
GenreCandidate signal: Review · Consensus signal: none
Teacher disagreement score0.005
Threshold uncertainty score0.011

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0020.004
Meta-epidemiology (narrow)0.0010.001
Meta-epidemiology (broad)0.0010.004
Bibliometrics0.0010.003
Science and technology studies0.0010.001
Scholarly communication0.0020.000
Open science0.0010.001
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0030.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.053
GPT teacher head0.347
Teacher spread0.294 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designSystematic review
Domainnot available
GenreReview

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations218
Published2014
Admission routes1
Has abstractyes

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