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Record W2045346543 · doi:10.1186/1897-4287-8-s1-p17

An unusual tumor spectrum in Lynch syndrome caused by MSH6 mutation

2010· article· en· W2045346543 on OpenAlexaff
Renée Perrier, Paulein Van Galen, Janice L. Pasieka, Tony Magliocco, A. Micheil Innes

Bibliographic record

VenueHereditary Cancer in Clinical Practice · 2010
Typearticle
Languageen
FieldMedicine
TopicGenetic factors in colorectal cancer
Canadian institutionsAlberta Children's Hospital
Fundersnot available
KeywordsMedicineLynch syndromeMSH6Family historyEndometrial cancerColorectal cancerCancer syndromeCancerInternal medicinePheochromocytomaNeurofibromatosisOncologyGermline mutationGastroenterologyPathologyDNA mismatch repairMutationGenetics

Abstract

fetched live from OpenAlex

Inherited cancer syndromes associated with acoustic neuroma (i.e. neurofibromatosis 2-NF2), pheochromocytoma (i.e. Von Hippel Lindau, NFl, multiple endocrine neoplasia syndromes, and hereditary paraganglioma syndrome), and colon cancer are well known. Lynch syndrome is the most common hereditary colon cancer syndrome and is caused by DNA mismatch repair dysfunction secondary to inherited mutations in one of MLHl, MSH2, MSH6, and less commonly PMS2. An increased risk for a variety of cancers is seen in patients with Lynch syndrome with the greatest risks being for colon and endometrial cancer. We report a Dutch patient with a history of bilateral acoustic neuromas diagnosed at 47, and pheochromocytoma and endometrial adenocarcinoma diagnosed at age 54. She had no family history or other signs/symptoms of NF2. Family history was significant for her brother having metachronous colon cancers at 42 and 51, and a maternal uncle having colon cancer in his 40s. The family does not fulfill either Amsterdam I or II criteria. Clinical investigations for hereditary cancer predisposition were undertaken in our patient given her history of multiple primary tumors. Immunohistochemistry (IHC) for MLH1, MSH2, and MSH6 proteins, and genetic testing for Lynch syndrome were completed. In light of the history of bilateral acoustic neuromas, genetic testing for NF2 was also undertaken. Genetic testing for NF2 did not detect a mutation or deletion in the NF2 gene. IHC on tissue from the patient's endometrial adenocarcinoma and pheochromocytoma showed absent expression of MSH6. A pathogenic germline mutation in MSH6 (c.651_652insT) was identified. We report a potential new association of Lynch syndrome with pheochromocytoma and acoustic neuromas in a woman with Lynch syndrome caused by an MSH6 mutation. In this case, a diagnosis of Lynch syndrome was suspected due the history of endometrial adenocarcinoma in our patient and her family history of early colon cancer. Identification of a Dutch founder mutation in MSH6 confirmed the diagnosis. The absence of MSH6 expression by IHC in both the endometrial carcinoma and the pheochromocytoma highly suggests an association of the pheochromocytoma with defective mismatch repair function secondary to the MSH6 mutation. Unfortunately, tumor studies could not be completed on tissue from the acoustic neuroma so it is difficult to say whether they arose independently or were also related to the diagnosis of Lynch syndrome. In this case, normal NF2 genetic testing and the absence of a family history of NF2 may suggest an association of acoustic neuromas with Lynch syndrome.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.002
metaresearch head score (Gemma)0.005
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow), Research integrity, Insufficient payload (model declined to judge)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.127
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0020.005
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0000.001
Science and technology studies0.0000.000
Scholarly communication0.0000.001
Open science0.0000.000
Research integrity0.0000.003
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.035
GPT teacher head0.420
Teacher spread0.385 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations3
Published2010
Admission routes1
Has abstractyes

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